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Publications by K Soós
Β-Amyloid(Phe(SO3H)24)25–35 in Rat Nucleus Basalis Induces Behavioral Dysfunctions, Impairs Learning and Memory and Disrupts Cortical Cholinergic Innervation
Behavioural Brain Research
Behavioral Neuroscience
Related publications
Cholinergic Fiber Aberrations in Nucleus Basalis Lesioned Rat and Alzheimer's Disease
Neurobiology of Aging
Aging
Gerontology
Developmental Biology
Geriatrics
Neuroscience
Neurology
Β-Amyloid Fragment 25–35 Causes Mitochondrial Dysfunction in Primary Cortical Neurons
Neurobiology of Disease
Neurology
Acute Β-Amyloid Administration Disrupts the Cholinergic Control of Dopamine Release in the Nucleus Accumbens
Neuropsychopharmacology
Psychiatry
Mental Health
Pharmacology
Amyloid Beta-Peptide Disrupts Carbachol-Induced Muscarinic Cholinergic Signal Transduction in Cortical Neurons.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Β-Amyloid 25-35 Peptide Reduces the Expression of Glutamine Transporter SAT1 in Cultured Cortical Neurons
Neurochemical Research
Medicine
Biochemistry
Cellular
Molecular Neuroscience
Amyloid Β-Peptide Directly Induces Spontaneous Calcium Transients, Delayed Intercellular Calcium Waves and Gliosis in Rat Cortical Astrocytes
ASN Neuro
Neuroscience
Neurology
Cholinergic Circuitry of the Human Nucleus Basalis and Its Fate in Alzheimer's Disease
Journal of Comparative Neurology
Neuroscience
Neonatal 6-Hydroxydopamine Treatment Eliminates Cholinergic Sympathetic Innervation and Induces Sensory Sprouting in Rat Sweat Glands
Journal of Neuroscience
Neuroscience
Amyloid-Β(25-35) Modulates the Expression of GirK and KCNQ Channel Genes in the Hippocampus
PLoS ONE
Multidisciplinary
