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Publications by K. Dezaki
Involvement of cAMP/EPAC/TRPM2 Activation in Glucose- And Incretin-Induced Insulin Secretion
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Related publications
GLUT2 and the Incretin Receptors Are Involved in Glucose-Induced Incretin Secretion
Molecular and Cellular Endocrinology
Biochemistry
Endocrinology
Molecular Biology
Glutamate Acts as a Key Signal Linking Glucose Metabolism to Incretin/cAMP Action to Amplify Insulin Secretion
Cell Reports
Biochemistry
Genetics
Molecular Biology
Targeting Development of Incretin-Producing Cells Increases Insulin Secretion
Journal of Clinical Investigation
Medicine
Stigmasterol Prevents Glucolipotoxicity Induced Defects in Glucose-Stimulated Insulin Secretion
Scientific Reports
Multidisciplinary
Glucose-Induced Insulin Secretion Independent of ATP-sensitive K+ Channels
Folia Pharmacologica Japonica
Pharmacology
Genetic Variants Affecting Incretin Sensitivity and Incretin Secretion
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Role of Epac2A/Rap1 Signaling in Interplay Between Incretin and Sulfonylurea in Insulin Secretion
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
The Regulation of Glucose-Induced Insulin Secretion by Pre-Stimulus Glucose Level and Tolbutamide in Normal Man
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
P2Y Purinergic Potentiation of Glucose-Induced Insulin Secretion and Pancreatic Beta-Cell Metabolism
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes