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Publications by Kathleen A. Kenner
Protein-Tyrosine Phosphatase 1B Is a Negative Regulator of Insulin- And Insulin-Like Growth Factor-I-Stimulated Signaling
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
Gαi2Enhances Insulin Signaling via Suppression of Protein-Tyrosine Phosphatase 1B
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Regulation of Insulin-Like Growth Factor Type I (IGF-I) Receptor Kinase Activity by Protein Tyrosine Phosphatase 1B (PTP-1B) and Enhanced IGF-I-Mediated Suppression of Apoptosis and Motility in PTP-1B-Deficient Fibroblasts
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Skeletal Muscle Protein Tyrosine Phosphatase Activity and Tyrosine Phosphatase 1B Protein Content Are Associated With Insulin Action and Resistance.
Journal of Clinical Investigation
Medicine
Insulin-Like Growth Factor-Binding Protein
Insulin Induces Heterologous Desensitization of G Protein-Coupled Receptor and Insulin-Like Growth Factor I Signaling by Downregulating -Arrestin-1
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Insulin-Like Growth Factor I and Insulin-Like Growth Factor Binding Protein 3 as Determinants of Blood Hemoglobin Concentration in Healthy Subjects
Pediatric Research
Child Health
Pediatrics
Perinatology
Insulin-Like Growth Factor-I-Induced DNA Synthesis in Insulin-Secreting Cell Line RINm5F Is Associated With Phosphorylation of the Insulin-Like Growth Factor-I Receptor and the Insulin Receptor Substrate-2
Journal of Endocrinology
Endocrinology
Metabolism
Diabetes
Insulin-Stimulated Hydrogen Peroxide Reversibly Inhibits Protein-Tyrosine Phosphatase 1Bin Vivoand Enhances the Early Insulin Action Cascade
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Protein Tyrosine Phosphatase 1B Is a Key Regulator of IFNAR1 Endocytosis and a Target for Antiviral Therapies
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary