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Publications by Katja Stifter
A Dominant Insulin-Specific and Islet-Destructive T-Cell Response Is Sufficient to Activate CD8 T Cells Directed Against the Fatty-Acid Receptor GPR40
Cellular and Molecular Immunology
Infectious Diseases
Medicine
Allergy
Immunology
Related publications
GPR40 Is Necessary but Not Sufficient for Fatty Acid Stimulation of Insulin Secretion in Vivo
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Altered Gut Microbiota Activate and Expand Insulin B15-23-Reactive CD8+ T-Cells
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
A Melanoma Multiepitope Polypeptide Induces Specific CD8+ T-Cell Response
Cellular Immunology
Immunology
Correction: Cross-Presentation of a Spread-Defective McMv Is Sufficient to Prime the Majority of Virus-Specific CD8+ T Cells
PLoS ONE
Multidisciplinary
Pathogen-Specific Inflammatory Milieux Tune the Antigen Sensitivity of CD8+ T Cells by Enhancing T Cell Receptor Signaling
Immunity
Infectious Diseases
Allergy
Immunology
GFP-specific CD8 T Cells Enable Targeted Cell Depletion and Visualization of T-Cell Interactions
Nature Biotechnology
Applied Microbiology
Biotechnology
Biomedical Engineering
Molecular Medicine
Bioengineering
Responses Against a Subdominant CD8+ T Cell Epitope Protect Against Immunopathology Caused by a Dominant Epitope
Journal of Immunology
Allergy
Immunology
Free Fatty Acid Receptor 1 (FFAR1; GPR40)
Science-Business eXchange
Structural Basis for Enabling T-Cell Receptor Diversity Within Biased Virus-Specific CD8+ T-Cell Responses
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary