Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
Publications by Keiichi Sakakibara
CBS9106-Induced CRM1 Degradation Is Mediated by Cullin Ring Ligase Activity and the Neddylation Pathway
Molecular Cancer Therapeutics
Cancer Research
Oncology
Related publications
Identification of the Degradation Determinants of Insulin Receptor Substrate 1 for Signaling Cullin-Ring E3 Ubiquitin Ligase 7-Mediated Ubiquitination
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Cullin E3 Ligase Activity Is Required for Myoblast Differentiation
Journal of Molecular Biology
Structural Biology
Molecular Biology
Biophysics
Adaptor Protein Self-Assembly Drives the Control of a Cullin-Ring Ubiquitin Ligase
Structure
Structural Biology
Molecular Biology
The Mechanism of Ubiquitination in the Cullin-Ring E3 Ligase Machinery: Conformational Control of Substrate Orientation
PLoS Computational Biology
Molecular Neuroscience
Evolution
Ecology
Genetics
Behavior
Molecular Biology
Systematics
Simulation
Cellular
Computational Theory
Mathematics
Modeling
Evidence for a Regulatory Role of Cullin-Ring E3 Ubiquitin Ligase 7 in Insulin Signaling
Cellular Signalling
Cell Biology
DHEA-induced Ovarian Hyperfibrosis Is Mediated by TGF-β Signaling Pathway
Journal of Ovarian Research
Gynecology
Oncology
Obstetrics
Regulation of PTEN Degradation and NEDD4–1 E3 Ligase Activity by Numb
Cell Cycle
Medicine
Developmental Biology
Cell Biology
Molecular Biology
The Cullin-Ring E3 Ubiquitin Ligase CRL4−DCAF1 Complex Dimerizes via a Short Helical Region in DCAF1
Biochemistry
Biochemistry
The Protein Neddylation Pathway inTrypanosoma Brucei
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology