Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Keith R. Anderson

MEKK-1, a Component of the Stress (Stress-Activated Protein Kinase/C-Jun N-Terminal Kinase) Pathway, Can Selectively Activate Smad2-Mediated Transcriptional Activation in Endothelial Cells

Journal of Biological Chemistry

English

Related publications

Activation of the C-Jun N-Terminal Kinase/Stress-Activated Protein Kinase Pathway by Overexpression of Caspase-8 and Its Homologs

Journal of Biological Chemistry

English

P57KIP2Modulates Stress-Activated Signaling by Inhibiting C-Jun NH2-terminal Kinase/Stress-Activated Protein Kinase

Journal of Biological Chemistry

English

Oxalate Selectively Activates P38 Mitogen-Activated Protein Kinase and C-Jun N-Terminal Kinase Signal Transduction Pathways in Renal Epithelial Cells

Journal of Biological Chemistry

English

Activation of the Hematopoietic Progenitor Kinase-1 (HPK1)-dependent, Stress-Activated C-Jun N-Terminal Kinase (JNK) Pathway by Transforming Growth Factor Β (TGF-β)-activated Kinase (TAK1), a Kinase Mediator of TGF Β Signal Transduction

Journal of Biological Chemistry

English

P21-Activated Protein Kinase 1 Induces the Invasion of Gastric Cancer Cells Through C-Jun NH2-terminal Kinase-Mediated Activation of Matrix Metalloproteinase-2

English

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

Journal of Biological Chemistry

English

C-Jun N-Terminal Kinase 1 (JNK1); VEGF

Science-Business eXchange

2009

English

Direct Activation of the Stress-Activated Protein Kinase (SAPK) and Extracellular Signal-Regulated Protein Kinase (ERK) Pathways by an Inducible Mitogen-Activated Protein Kinase/Erk Kinase Kinase 3 (MEKK) Derivative

Journal of Biological Chemistry

English

Fluid Shear Stress Activation of Egr-1 Transcription in Cultured Human Endothelial and Epithelial Cells Is Mediated via the Extracellular Signal-Related Kinase 1/2 Mitogen-Activated Protein Kinase Pathway.

Journal of Clinical Investigation

Medicine

1998

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Keith R. Anderson

MEKK-1, a Component of the Stress (Stress-Activated Protein Kinase/C-Jun N-Terminal Kinase) Pathway, Can Selectively Activate Smad2-Mediated Transcriptional Activation in Endothelial Cells

Related publications

Activation of the C-Jun N-Terminal Kinase/Stress-Activated Protein Kinase Pathway by Overexpression of Caspase-8 and Its Homologs

P57KIP2Modulates Stress-Activated Signaling by Inhibiting C-Jun NH2-terminal Kinase/Stress-Activated Protein Kinase

Oxalate Selectively Activates P38 Mitogen-Activated Protein Kinase and C-Jun N-Terminal Kinase Signal Transduction Pathways in Renal Epithelial Cells

Activation of the Hematopoietic Progenitor Kinase-1 (HPK1)-dependent, Stress-Activated C-Jun N-Terminal Kinase (JNK) Pathway by Transforming Growth Factor Β (TGF-β)-activated Kinase (TAK1), a Kinase Mediator of TGF Β Signal Transduction

P21-Activated Protein Kinase 1 Induces the Invasion of Gastric Cancer Cells Through C-Jun NH2-terminal Kinase-Mediated Activation of Matrix Metalloproteinase-2

GqProtein-induced Apoptosis Is Mediated by AKT Kinase Inhibition That Leads to Protein Kinase C-Induced C-Jun N-Terminal Kinase Activation

C-Jun N-Terminal Kinase 1 (JNK1); VEGF

Direct Activation of the Stress-Activated Protein Kinase (SAPK) and Extracellular Signal-Regulated Protein Kinase (ERK) Pathways by an Inducible Mitogen-Activated Protein Kinase/Erk Kinase Kinase 3 (MEKK) Derivative

Fluid Shear Stress Activation of Egr-1 Transcription in Cultured Human Endothelial and Epithelial Cells Is Mediated via the Extracellular Signal-Related Kinase 1/2 Mitogen-Activated Protein Kinase Pathway.