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Publications by Kristine Freude
Neurons Derived From Sporadic Alzheimer’s Disease iPSCs Reveal Elevated TAU Hyperphosphorylation, Increased Amyloid Levels, and GSK3B Activation
Alzheimer's Research and Therapy
Neurology
Cognitive Neuroscience
Related publications
Familial Alzheimer’s Disease Patient-Derived Neurons Reveal Distinct Mutation-Specific Effects on Amyloid Beta
Molecular Psychiatry
Psychiatry
Molecular Neuroscience
Mental Health
Molecular Biology
Cellular
Tau Filaments From Multiple Cases of Sporadic and Inherited Alzheimer’s Disease Adopt a Common Fold
Acta Neuropathologica
Forensic Medicine
Molecular Neuroscience
Pathology
Neurology
Cellular
Activation of Asparaginyl Endopeptidase Leads to Tau Hyperphosphorylation in Alzheimer Disease
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Diagnosis of Alzheimer’s Disease Utilizing Amyloid and Tau as Fluid Biomarkers
Experimental and Molecular Medicine
Biochemistry
Medicine
Clinical Biochemistry
Molecular Biology
Molecular Medicine
Reductions in Amyloid-Β-Derived Neuroinflammation, With Minocycline, Restore Cognition but Do Not Significantly Affect Tau Hyperphosphorylation
Journal of Alzheimer's Disease
Gerontology
Clinical Psychology
Mental Health
Psychiatry
Geriatrics
Medicine
Neuroscience
The Spleen Tyrosine Kinase (Syk) Regulates Alzheimer Amyloid-Β Production and Tau Hyperphosphorylation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Hyperphosphorylation-Induced Tau Oligomers
Frontiers in Neurology
Neurology
GWAS of Cerebrospinal Fluid Tau Levels Identifies Risk Variants for Alzheimer’s Disease
Neuron
Neuroscience
Loss of Hsp110 Leads to Age-Dependent Tau Hyperphosphorylation and Early Accumulation of Insoluble Amyloid
Molecular and Cellular Biology
Cell Biology
Molecular Biology
