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Publications by Kyle D. Copps
Serine 302 Phosphorylation of Mouse Insulin Receptor Substrate 1 (IRS1) Is Dispensable for Normal Insulin Signaling and Feedback Regulation by Hepatic S6 Kinase
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
Regulation of Insulin Sensitivity by Serine/Threonine Phosphorylation of Insulin Receptor Substrate Proteins IRS1 and IRS2
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Tyrosine Phosphorylation of Phosphoinositide-Dependent Kinase 1 by the Insulin Receptor Is Necessary for Insulin Metabolic Signaling
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Insulin Regulation of Membrane-Associated Insulin Receptor Substrate 1
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Insulin-Induced Serine 22 Phosphorylation of Retinoid X Receptor Alpha Is Dispensable for Adipogenesis in Brown Adipocytes
Adipocyte
Histology
Cell Biology
Insulin Stimulates PKCζ-mediated Phosphorylation of Insulin Receptor Substrate-1 (IRS-1)
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Phosphatidylinositol 3′-Kinase Associates With an Insulin Receptor Substrate-1 Serine Kinase Distinct From Its Intrinsic Serine Kinase
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Hepatic Insulin Signalling Is Dispensable for Suppression of Glucose Output by Insulin in Vivo
Nature Communications
Astronomy
Genetics
Molecular Biology
Biochemistry
Chemistry
Physics
Modulation of Insulin Receptor Substrate-1 Tyrosine Phosphorylation by an Akt/Phosphatidylinositol 3-Kinase Pathway
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Prolactin Activates Tyrosyl Phosphorylation of Insulin Receptor Substrate 1 and Phosphatidylinositol-3-Oh Kinase
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology