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Publications by L. Lanier
Nonfunctional Mutants of the Retinoblastoma Protein Are Characterized by Defects in Phosphorylation, Viral Oncoprotein Association, and Nuclear Tethering.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Related publications
Dysferlin Mutants: Defects in Trafficking and Association With Proteins of the Transverse Tubule
Biophysical Journal
Biophysics
Nuclear Protein Phosphorylation and Growth Control
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Nuclear Import of LASP-1 Is Regulated by Phosphorylation and Dynamic Protein–protein Interactions
Oncogene
Cancer Research
Genetics
Molecular Biology
The High-Risk HPV16 E7 Oncoprotein Mediates Interaction Between the Transcriptional Coactivator CBP and the Retinoblastoma Protein pRb
Journal of Molecular Biology
Structural Biology
Molecular Biology
Biophysics
PITALRE, a Nuclear CDC2-related Protein Kinase That Phosphorylates the Retinoblastoma Protein in Vitro.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Limited Redundancy in Phosphorylation of Retinoblastoma Tumor Suppressor Protein by Cyclin-Dependent Kinases in Acute Lymphoblastic Leukemia
American Journal of Pathology
Forensic Medicine
Pathology
Mutants of Herpes Simplex Virus. I. Isolation and Properties of Mutants Characterized by Plaque Size and Cytopathogenicity
Kurume Medical Journal
Medicine
Learning Defects in Drosophila Growth Restricted Chico Mutants Are Caused by Attenuated Adenylyl Cyclase Activity
Molecular Brain
Molecular Neuroscience
Molecular Biology
Cellular
Zebrafish Sin3b Mutants Are Viable but Have Size, Skeletal and Locomotor Defects
Developmental Dynamics
Developmental Biology
