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Publications by L. Mucke

Neurotoxicity of Amyloid -Protein: Synaptic and Network Dysfunction

Cold Spring Harbor perspectives in medicine
BiochemistryMedicineGeneticsMolecular Biology
2012English

-Amyloid Peptides Enhance -Synuclein Accumulation and Neuronal Deficits in a Transgenic Mouse Model Linking Alzheimer's Disease and Parkinson's Disease

Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
2001English

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Endophilin 1 Knockdown Prevents Synaptic Dysfunction Induced by Oligomeric Amyloid Β

Molecular Medicine Reports
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2019English

Alzheimer's Disease Risk Factor Pyk2 Mediates Amyloid-Β Induced Synaptic Dysfunction and Loss

Journal of Neuroscience
Neuroscience
2018English

Catecholamines Potentiate Amyloid Β-Peptide Neurotoxicity: Involvement of Oxidative Stress, Mitochondrial Dysfunction, and Perturbed Calcium Homeostasis

Neurobiology of Disease
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1998English

Synaptic Dysfunction and Septin Protein Family Members in Neurodegenerative Diseases

Molecular Neurodegeneration
Molecular NeuroscienceNeurologyMolecular BiologyCellular
2015English

Intracellular Accumulation of Amyloid-Beta – A Predictor for Synaptic Dysfunction and Neuron Loss in Alzheimer’s Disease

Frontiers in Aging Neuroscience
AgingCognitive Neuroscience
2010English

Amyloid-Β Protein Oligomer at Low Nanomolar Concentrations Activates Microglia and Induces Microglial Neurotoxicity

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2010English

Iron Enhances the Neurotoxicity of Amyloid Β

Translational Stroke Research
NeuroscienceNeurologyCardiovascular MedicineCardiology
2011English

Amyloid Beta, Mitochondrial Dysfunction and Synaptic Damage: Implications for Cognitive Decline in Aging and Alzheimer's Disease

Trends in Molecular Medicine
Molecular MedicineMolecular Biology
2008English

Protofibrillar Intermediates of Amyloid Β-Protein Induce Acute Electrophysiological Changes and Progressive Neurotoxicity in Cortical Neurons

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