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Publications by Lingpeng Pei
Clomipramine Causes Osteoporosis by Promoting Osteoclastogenesis via E3 Ligase Itch, Which Is Prevented by Zoledronic Acid
Scientific Reports
Multidisciplinary
Related publications
Human ITCH E3 Ubiquitin Ligase Deficiency Causes Syndromic Multisystem Autoimmune Disease
American Journal of Human Genetics
Genetics
The E3 Ubiquitin Ligase Itch Is Required for B-Cell Development
Scientific Reports
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Ubiquitin E3 Ligase Itch Negatively Regulates Osteoclast Formation by Promoting Deubiquitination of Tumor Necrosis Factor (TNF) Receptor-Associated Factor 6
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
TRIAD1 Is Negatively Regulated by the MDM2 E3 Ligase
Oncology Reports
Medicine
Cancer Research
Oncology
TIN2 Stability Is Regulated by the E3 Ligase Siah2
Molecular and Cellular Biology
Cell Biology
Molecular Biology
E3 Ligase FBXW7 Aggravates TMPD-induced Systemic Lupus Erythematosus by Promoting Cell Apoptosis
Cellular and Molecular Immunology
Infectious Diseases
Medicine
Allergy
Immunology
The Ubiquitin E3 Ligase ITCH Enhances Breast Tumor Progression by Inhibiting the Hippo Tumor Suppressor Pathway
Oncotarget
Oncology
Rain Focal Ischemia-Reperfusion Causes a Decreased Resistance of Еrythrocytes From Venous Blood to Acid Hemolysis, Which Is Prevented by Ecdysterone
Fiziolohichnyĭ zhurnal
The Nedd4-Binding Partner 1 (N4BP1) Protein Is an Inhibitor of the E3 Ligase Itch
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
