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Publications by Louis M. Luttrell

The Arrestin-Selective Angiotensin AT1Receptor Agonist [Sar1,Ile4,Ile8]-AngII Negatively Regulates Bradykinin B2Receptor Signaling via AT1-B2Receptor Heterodimers

Journal of Biological Chemistry

English

HDL3, but Not HDL2, Stimulates Plasminogen Activator Inhibitor-1 Release From Adipocytes: The Role of Sphingosine-1-Phosphate

Journal of Lipid Research

English

Informatic Deconvolution of Biased GPCR Signaling Mechanisms From in Vivo Pharmacological Experimentation

English

A High-Content, Live-Cell, and Real-Time Approach to the Quantitation of Ligand-Induced Β-Arrestin2 and Class a/Class B GPCR Mobilization

Microscopy and Microanalysis

Instrumentation

2013

English

SRC-Dependent Tyrosine Phosphorylation Regulates Dynamin Self-Assembly and Ligand-Induced Endocytosis of the Epidermal Growth Factor Receptor

Journal of Biological Chemistry

English

GβγSubunits Mediate Mitogen-Activated Protein Kinase Activation by the Tyrosine Kinase Insulin-Like Growth Factor 1 Receptor

Journal of Biological Chemistry

English

Arrestin-Mediated ERK Activation by Gonadotropin-Releasing Hormone Receptors

Journal of Biological Chemistry

English

Protein Kinase a and G Protein-Coupled Receptor Kinase Phosphorylation Mediates Β-1 Adrenergic Receptor Endocytosis Through Different Pathways

Journal of Biological Chemistry

English

Β-Arrestins 1 and 2 Differentially Regulate LPS-induced Signaling and Pro-Inflammatory Gene Expression

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Louis M. Luttrell

The Arrestin-Selective Angiotensin AT1Receptor Agonist [Sar1,Ile4,Ile8]-AngII Negatively Regulates Bradykinin B2Receptor Signaling via AT1-B2Receptor Heterodimers

HDL3, but Not HDL2, Stimulates Plasminogen Activator Inhibitor-1 Release From Adipocytes: The Role of Sphingosine-1-Phosphate

Informatic Deconvolution of Biased GPCR Signaling Mechanisms From in Vivo Pharmacological Experimentation

A High-Content, Live-Cell, and Real-Time Approach to the Quantitation of Ligand-Induced Β-Arrestin2 and Class a/Class B GPCR Mobilization

SRC-Dependent Tyrosine Phosphorylation Regulates Dynamin Self-Assembly and Ligand-Induced Endocytosis of the Epidermal Growth Factor Receptor

GβγSubunits Mediate Mitogen-Activated Protein Kinase Activation by the Tyrosine Kinase Insulin-Like Growth Factor 1 Receptor

Arrestin-Mediated ERK Activation by Gonadotropin-Releasing Hormone Receptors

Protein Kinase a and G Protein-Coupled Receptor Kinase Phosphorylation Mediates Β-1 Adrenergic Receptor Endocytosis Through Different Pathways

Β-Arrestins 1 and 2 Differentially Regulate LPS-induced Signaling and Pro-Inflammatory Gene Expression