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Publications by Ludek Cervenka
Effects of Angiotensin II AT1^|^ndash;Receptor Blockade on High Fat Diet^|^ndash;Induced Vascular Oxidative Stress and Endothelial Dysfunction in Dahl Salt-Sensitive Rats
Journal of Pharmacological Sciences
Molecular Medicine
Pharmacology
Proximal Tubular Angiotensin II Levels and Renal Functional Responses to AT1 Receptor Blockade in Nonclipped Kidneys of Goldblatt Hypertensive Rats
Hypertension
Internal Medicine
Related publications
Effects of AT1 Receptor Blockade on Renal Injury and Mitogen-Activated Protein Activity in Dahl Salt-Sensitive Rats
Kidney International
Nephrology
Angiotensin II (AT1) Receptor Blockade Reduces Vascular Tissue Factor in Angiotensin II-Induced Cardiac Vasculopathy
American Journal of Pathology
Forensic Medicine
Pathology
Effect of AT1 Receptor Blockade on Intermittent Hypoxia-Induced Endothelial Dysfunction
Respiratory Physiology and Neurobiology
Neuroscience
Pulmonary
Respiratory Medicine
Physiology
Effect of AT1 Receptor Blockade on Expression of Genes for Type-1 Angiotensin II(AT1) Receptor Subtypes in Spontaneously Hypertensive Rats.
Hypertension Research
Internal Medicine
Cardiovascular Medicine
Physiology
Cardiology
ETA Receptor Blockade Prevents Increased Tissue Endothelin-1, Vascular Hypertrophy, and Endothelial Dysfunction in Salt-Sensitive Hypertension
Hypertension
Internal Medicine
Effects of Sesamin on Aortic Oxidative Stress and Endothelial Dysfunction in Deoxycorticosterone Acetate-Salt Hypertensive Rats
Biological and Pharmaceutical Bulletin
Medicine
Pharmacology
Pharmaceutical Science
Genetic Linkage of Albuminuria and Renal Injury in Dahl Salt-Sensitive Rats on a High-Salt Diet: Comparison With Spontaneously Hypertensive Rats
Physiological Genomics
Genetics
Physiology
Angiotensin 1-7 Protects Against Angiotensin II-Induced Endoplasmic Reticulum Stress and Endothelial Dysfunction via Mas Receptor
PLoS ONE
Multidisciplinary
Atorvastatin Prevents Angiotensin II-Induced Vascular Remodeling and Oxidative Stress
Hypertension
Internal Medicine
