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Publications by M S Sy

Genetic Restrictions for the Induction of Suppressor T Cells by Hapten- Modified Lymphoid Cells in Tolerance to 1-Fluoro-2,4-Dinitrobenzene Contact Sensitivity. Role of the H-2d Region of the Major Histocompatibility Complex

Journal of Experimental Medicine

English

Antigen- And Receptor-Driven Regulatory Mechanisms. II. Induction of Suppressor T Cells With Idiotype-Coupled Syngeneic Spleen Cells

Journal of Experimental Medicine

English

Related publications

T Cells Specific for Hapten-Modified Self Are Precommitted for Self Major Histocompatibility Complex Antigens Before Encounter With the Hapten

Journal of Experimental Medicine

English

A Role for Suppressor T Cells in Induction of Self-Tolerance.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1985

English

Tolerance to Parenchymal Self. Regulatory Role of Major Histocompatibility Complex-Restricted, OX8+ Suppressor T Cells Specific for Autologous Renal Tubular Antigen in Experimental Interstitial Nephritis

Journal of Experimental Medicine

English

Presentation of a Self-Peptide for in Vivo Tolerance Induction of CD4+ T Cells Is Governed by a Processing Factor That Maps to the Class II Region of the Major Histocompatibility Complex Locus

Journal of Experimental Medicine

English

Mechanisms of Tolerance Induction in Major Histocompatibility Complex Class II-restricted T Cells Specific for a Blood-Borne Self-Antigen

Journal of Experimental Medicine

English

HLA Antigens Expressed on Murine Cells Are Preferentially Recognized by Murine Cytotoxic T Cells in the Context of the H-2 Major Histocompatibility Complex.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1986

English

T Cell Regulation of B Cell Activation. I-A-Restricted T Suppressor Cells Inhibit the Major Histocompatibility Complex-Restricted Interactions of T Helper Cells With B Cells and Accessory Cells

Journal of Experimental Medicine

English

Major Histocompatibility Complex Class II-associated Peptides Control the Presentation of Bacterial Superantigens to T Cells

Journal of Experimental Medicine

English

Joint Recognition by Cytotoxic T Cells of Inactivated Sendai Virus and Products of the Major Histocompatibility Complex

Journal of Experimental Medicine

English

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Discover open access scientific publications

Search, annotate, share and cite publications

Publications by M S Sy

Genetic Restrictions for the Induction of Suppressor T Cells by Hapten- Modified Lymphoid Cells in Tolerance to 1-Fluoro-2,4-Dinitrobenzene Contact Sensitivity. Role of the H-2d Region of the Major Histocompatibility Complex

Antigen- And Receptor-Driven Regulatory Mechanisms. II. Induction of Suppressor T Cells With Idiotype-Coupled Syngeneic Spleen Cells

Related publications

T Cells Specific for Hapten-Modified Self Are Precommitted for Self Major Histocompatibility Complex Antigens Before Encounter With the Hapten

A Role for Suppressor T Cells in Induction of Self-Tolerance.

Tolerance to Parenchymal Self. Regulatory Role of Major Histocompatibility Complex-Restricted, OX8+ Suppressor T Cells Specific for Autologous Renal Tubular Antigen in Experimental Interstitial Nephritis

Presentation of a Self-Peptide for in Vivo Tolerance Induction of CD4+ T Cells Is Governed by a Processing Factor That Maps to the Class II Region of the Major Histocompatibility Complex Locus

Mechanisms of Tolerance Induction in Major Histocompatibility Complex Class II-restricted T Cells Specific for a Blood-Borne Self-Antigen

HLA Antigens Expressed on Murine Cells Are Preferentially Recognized by Murine Cytotoxic T Cells in the Context of the H-2 Major Histocompatibility Complex.

T Cell Regulation of B Cell Activation. I-A-Restricted T Suppressor Cells Inhibit the Major Histocompatibility Complex-Restricted Interactions of T Helper Cells With B Cells and Accessory Cells

Major Histocompatibility Complex Class II-associated Peptides Control the Presentation of Bacterial Superantigens to T Cells

Joint Recognition by Cytotoxic T Cells of Inactivated Sendai Virus and Products of the Major Histocompatibility Complex