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Publications by M Seman

Possible Role of the V3 Domain of Gp120 in Resistance to an Amphotericin B Derivative (MS8209) Blocking Human Immunodeficiency Virus Entry.

Journal of Virology

English

Amphotericin B Derivative Blocks Human Immunodeficiency Virus Type 1 Entry After CD4 Binding: Effect on Virus-Cell Fusion but Not on Cell-Cell Fusion.

English

Related publications

Maternofetal Transmission of Human Immunodeficiency Virus-1: The Role of Antibodies to the V3 Primary Neutralizing Domain

English

Anti-Human Immunodeficiency Virus Type 1 Activity of an Oligocationic Compound Mediated via Gp120 V3 Interactions.

English

Localized Changes in the Gp120 Envelope Glycoprotein Confer Resistance to Human Immunodeficiency Virus Entry Inhibitors BMS-806 and #155

English

Role of CD4 Epitopes Outside the Gp120-Binding Site During Entry of Human Immunodeficiency Virus Type 1.

English

The Human Immunodeficiency Virus Type 1 Gp120 V2 Domain Mediates Gp41-Independent Intersubunit Contacts

English

Sensitivity of Human Immunodeficiency Virus Type 1 to the Fusion Inhibitor T-20 Is Modulated by Coreceptor Specificity Defined by the V3 Loop of Gp120

English

N-Butyldeoxynojirimycin-Mediated Inhibition of Human Immunodeficiency Virus Entry Correlates With Impaired Gp120 Shedding and Gp41 Exposure.

English

Convergent Evolution Within the V3 Loop Domain of Human Immunodeficiency Virus Type 1 in Association With Disease Progression.

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by M Seman

Possible Role of the V3 Domain of Gp120 in Resistance to an Amphotericin B Derivative (MS8209) Blocking Human Immunodeficiency Virus Entry.

Amphotericin B Derivative Blocks Human Immunodeficiency Virus Type 1 Entry After CD4 Binding: Effect on Virus-Cell Fusion but Not on Cell-Cell Fusion.

Related publications

Maternofetal Transmission of Human Immunodeficiency Virus-1: The Role of Antibodies to the V3 Primary Neutralizing Domain

Anti-Human Immunodeficiency Virus Type 1 Activity of an Oligocationic Compound Mediated via Gp120 V3 Interactions.

Localized Changes in the Gp120 Envelope Glycoprotein Confer Resistance to Human Immunodeficiency Virus Entry Inhibitors BMS-806 and #155

Role of CD4 Epitopes Outside the Gp120-Binding Site During Entry of Human Immunodeficiency Virus Type 1.

The Human Immunodeficiency Virus Type 1 Gp120 V2 Domain Mediates Gp41-Independent Intersubunit Contacts

Sensitivity of Human Immunodeficiency Virus Type 1 to the Fusion Inhibitor T-20 Is Modulated by Coreceptor Specificity Defined by the V3 Loop of Gp120

N-Butyldeoxynojirimycin-Mediated Inhibition of Human Immunodeficiency Virus Entry Correlates With Impaired Gp120 Shedding and Gp41 Exposure.

Convergent Evolution Within the V3 Loop Domain of Human Immunodeficiency Virus Type 1 in Association With Disease Progression.