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Publications by M. Erta
Trans-Signaling Is a Dominant Mechanism for the Pathogenic Actions of Interleukin-6 in the Brain
Journal of Neuroscience
Neuroscience
Related publications
Alternative Intronic Polyadenylation Generates the Interleukin-6 Trans-Signaling Inhibitor Sgp130-E10
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Targeting Interleukin-6 Trans-Signaling in Head and Neck Squamous Cell Carcinoma
Trans-Presentation of IL-6 by Dendritic Cells Is Required for the Priming of Pathogenic TH17 Cells
Nature Immunology
Allergy
Immunology
Although IL-6 Trans-Signaling Is Sufficient to Drive Local Immune Responses, Classical IL-6 Signaling Is Obligate for the Induction of T Cell-Mediated Autoimmunity
Journal of Immunology
Allergy
Immunology
Activation of the Stat3 Signaling Pathway Is Required for Differentiation by Interleukin-6 in PC12-E2 Cells
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Transforming Growth Factor-Β Dampens Interleukin-6 Signaling in Chondrocytes by Decreasing the Interleukin-6 Receptor
Osteoarthritis and Cartilage
Sports Medicine
Rheumatology
Orthopedics
Biomedical Engineering
Activity-Dependent Brain-Derived Neurotrophic Factor Signaling Is Required for the Antidepressant Actions of (2r,6r)-Hydroxynorketamine
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Autocrine Production of Interleukin-6: A Mechanism of Interleukin-6 Independence in Dexamethasone-Resistant 7TD1 Murine Myeloma Cells
Journal of Cancer Therapy
N-Linked Glycosylation Is Essential for the Stability but Not the Signaling Function of the Interleukin-6 Signal Transducer Glycoprotein 130
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
