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Publications by M. Hyatt

Maternal Nutrient Restriction Between Early-To-Mid Gestation Amplifies the Insulin Response to Obesity in Conjunction With Increased mRNA Abundance for GLUT4 but Not the Insulin Receptor in Skeletal Muscle

Proceedings of the Nutrition Society

English

Related publications

Monoclonal Antibodies to the Human Insulin Receptor That Activate Glucose Transport but Not Insulin Receptor Kinase Activity.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1987

English

Hepatic Response to Restoration of GLUT4 in Skeletal Muscle of GLUT4 Null Mice

American Journal of Physiology - Endocrinology and Metabolism

English

Erratum To: Reduced AMP-activated Protein Kinase Activity in Mouse Skeletal Muscle Does Not Exacerbate the Development of Insulin Resistance With Obesity

English

Deletion of Skeletal Muscle SOCS3 Prevents Insulin Resistance in Obesity

English

Monoclonal Antibodies to the Insulin Receptor Mimic Metabolic Effects of Insulin but Do Not Stimulate Receptor Autophosphorylation in Transfected NIH 3T3 Fibroblasts.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1989

English

Mechanism of Increased Risk of Insulin Resistance in Aging Skeletal Muscle

Diabetology and Metabolic Syndrome

English

The Transcriptional Response to Oxidative Stress Is Part Of, but Not Sufficient For, Insulin Resistance in Adipocytes

Scientific Reports

Multidisciplinary

2018

English

Mechanistic Explanations for Counter-Intuitive Phosphorylation Dynamics of the Insulin Receptor and Insulin Receptor Substrate-1 in Response to Insulin in Murine Adipocytes

English

Reduced AMP-activated Protein Kinase Activity in Mouse Skeletal Muscle Does Not Exacerbate the Development of Insulin Resistance With Obesity

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by M. Hyatt

Maternal Nutrient Restriction Between Early-To-Mid Gestation Amplifies the Insulin Response to Obesity in Conjunction With Increased mRNA Abundance for GLUT4 but Not the Insulin Receptor in Skeletal Muscle

Related publications

Monoclonal Antibodies to the Human Insulin Receptor That Activate Glucose Transport but Not Insulin Receptor Kinase Activity.

Hepatic Response to Restoration of GLUT4 in Skeletal Muscle of GLUT4 Null Mice

Erratum To: Reduced AMP-activated Protein Kinase Activity in Mouse Skeletal Muscle Does Not Exacerbate the Development of Insulin Resistance With Obesity

Deletion of Skeletal Muscle SOCS3 Prevents Insulin Resistance in Obesity

Monoclonal Antibodies to the Insulin Receptor Mimic Metabolic Effects of Insulin but Do Not Stimulate Receptor Autophosphorylation in Transfected NIH 3T3 Fibroblasts.

Mechanism of Increased Risk of Insulin Resistance in Aging Skeletal Muscle

The Transcriptional Response to Oxidative Stress Is Part Of, but Not Sufficient For, Insulin Resistance in Adipocytes

Mechanistic Explanations for Counter-Intuitive Phosphorylation Dynamics of the Insulin Receptor and Insulin Receptor Substrate-1 in Response to Insulin in Murine Adipocytes

Reduced AMP-activated Protein Kinase Activity in Mouse Skeletal Muscle Does Not Exacerbate the Development of Insulin Resistance With Obesity