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Publications by M. Schuchmann
Histone Deacetylase Inhibition by Valproic Acid Down-Regulates C-Flip/Cash and Sensitizes Hepatoma Cells Towards CD95- And TRAIL Receptor-Mediated Apoptosis and Chemotherapy
Oncology Reports
Medicine
Cancer Research
Oncology
Related publications
Oxidative Stress Sensitizes Bladder Cancer Cells to TRAIL Mediated Apoptosis by Down-Regulating Anti-Apoptotic Proteins
Journal of Urology
Urology
Histone Deacetylase Inhibitors Valproic Acid and Vorinostat Enhance Trastuzumab-Mediated Antibody-Dependent Cell-Mediated Phagocytosis
Journal for ImmunoTherapy of Cancer
Oncology
Molecular Medicine
Pharmacology
Cancer Research
Allergy
Immunology
Histone Deacetylase Is a Target of Valproic Acid-Mediated Cellular Differentiation
Cancer Research
Cancer Research
Oncology
MUC16 Mucin (CA125) Attenuates TRAIL-induced Apoptosis by Decreasing TRAIL Receptor R2 Expression and Increasing C-Flip Expression
BMC Cancer
Cancer Research
Oncology
Genetics
Bufalin Sensitizes Human Bladder Carcinoma Cells to TRAIL-mediated Apoptosis
Oncology Letters
Cancer Research
Oncology
Luteolin Sensitizes Human Liver Cancer Cells to TRAIL‑induced Apoptosis via Autophagy and JNK‑mediated Death Receptor 5 Upregulation
International Journal of Oncology
Cancer Research
Oncology
Histone Deacetylase Inhibitors Valproic Acid and Depsipeptide Sensitize Retinoblastoma Cells to Radiotherapy by Increasing H2AX Phosphorylation and P53 Acetylation-Phosphorylation
International Journal of Oncology
Cancer Research
Oncology
The Contact Allergen Nickel Sensitizes Primary Human Endothelial Cells and Keratinocytes to TRAIL-mediated Apoptosis
Journal of Cellular and Molecular Medicine
Molecular Medicine
Cell Biology
Histone Deacetylase Inhibitor Valproic Acid Suppresses the Growth and Increases the Androgen Responsiveness of Prostate Cancer Cells
Cancer Letters
Cancer Research
Oncology