Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
Publications by M.A. Sharpe
Β-Amyloid Fragment 25–35 Causes Mitochondrial Dysfunction in Primary Cortical Neurons
Neurobiology of Disease
Neurology
Related publications
Β-Amyloid 25-35 Peptide Reduces the Expression of Glutamine Transporter SAT1 in Cultured Cortical Neurons
Neurochemical Research
Medicine
Biochemistry
Cellular
Molecular Neuroscience
TDP-43 Causes Neurotoxicity and Cytoskeletal Dysfunction in Primary Cortical Neurons
PLoS ONE
Multidisciplinary
Mitochondrial Dysfunction Causes Hyperexcitability of Serotonergic Neurons
Molecular Psychiatry
Psychiatry
Molecular Neuroscience
Mental Health
Molecular Biology
Cellular
Benchmarking Implicit Solvent Folding Simulations of the Amyloid Β(10−35) Fragment†
Journal of Physical Chemistry B
Surfaces
Theoretical Chemistry
Materials Chemistry
Films
Medicine
Coatings
Physical
Caspase-Cleaved Tau Expression Induces Mitochondrial Dysfunction in Immortalized Cortical Neurons
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Amyloid-Β Peptide Induces Mitochondrial Dysfunction by Inhibition of Preprotein Maturation
Cell Metabolism
Cell Biology
Molecular Biology
Physiology
Β-Amyloid(Phe(SO3H)24)25–35 in Rat Nucleus Basalis Induces Behavioral Dysfunctions, Impairs Learning and Memory and Disrupts Cortical Cholinergic Innervation
Behavioural Brain Research
Behavioral Neuroscience
Amyloid-Β Causes Memory Impairment by Disturbing the JAK2/STAT3 Axis in Hippocampal Neurons
Molecular Psychiatry
Psychiatry
Molecular Neuroscience
Mental Health
Molecular Biology
Cellular
Amyloid-Β(25-35) Modulates the Expression of GirK and KCNQ Channel Genes in the Hippocampus
PLoS ONE
Multidisciplinary
