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Publications by MENG WEI

GRIM-19-mediated Stat3 Activation Is a Determinant for Resveratrol-Induced Proliferation and Cytotoxicity in Cervical Tumor-Derived Cell Lines

Molecular Medicine Reports

English

Related publications

High Glucose Induces Down-Regulated GRIM-19 Expression to Activate STAT3 Signaling and Promote Cell Proliferation in Cell Culture

PLoS ONE

Multidisciplinary

2016

English

Tumor Suppressor P16INK4A Is Necessary for Survival of Cervical Carcinoma Cell Lines

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2013

English

GRIM-19: A Master Regulator of Cytokine Induced Tumor Suppression, Metastasis and Energy Metabolism

Cytokine and Growth Factor Reviews

English

Carbon Nanoparticle-Induced Lung Epithelial Cell Proliferation Is Mediated by Receptor-Dependent Akt Activation

American Journal of Physiology - Lung Cellular and Molecular Physiology

English

GRIM-19 Over-Expression Represses the Proliferation and Invasion of Orthotopically Implanted Hepatocarcinoma Tumors Associated With Downregulation of Stat3 Signaling

English

Survivin Suppression Through STAT3/β-catenin Is Essential for Resveratrol-Induced Melanoma Apoptosis

International Journal of Oncology

Cancer Research

Oncology

2014

English

Regulation of Cell-Mediated Cytotoxicity: I. Augmentation of Cell-Mediated Cytotoxicity Induced by Radiation

Journal of Experimental Medicine

English

Betacellulin-Induced Beta Cell Proliferation and Regeneration Is Mediated by Activation of ErbB-1 and ErbB-2 Receptors

PLoS ONE

Multidisciplinary

2011

English

Oncostatin M Promotes STAT3 Activation, VEGF Production, and Invasion in Osteosarcoma Cell Lines

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by MENG WEI

GRIM-19-mediated Stat3 Activation Is a Determinant for Resveratrol-Induced Proliferation and Cytotoxicity in Cervical Tumor-Derived Cell Lines

Related publications

High Glucose Induces Down-Regulated GRIM-19 Expression to Activate STAT3 Signaling and Promote Cell Proliferation in Cell Culture

Tumor Suppressor P16INK4A Is Necessary for Survival of Cervical Carcinoma Cell Lines

GRIM-19: A Master Regulator of Cytokine Induced Tumor Suppression, Metastasis and Energy Metabolism

Carbon Nanoparticle-Induced Lung Epithelial Cell Proliferation Is Mediated by Receptor-Dependent Akt Activation

GRIM-19 Over-Expression Represses the Proliferation and Invasion of Orthotopically Implanted Hepatocarcinoma Tumors Associated With Downregulation of Stat3 Signaling

Survivin Suppression Through STAT3/β-catenin Is Essential for Resveratrol-Induced Melanoma Apoptosis

Regulation of Cell-Mediated Cytotoxicity: I. Augmentation of Cell-Mediated Cytotoxicity Induced by Radiation

Betacellulin-Induced Beta Cell Proliferation and Regeneration Is Mediated by Activation of ErbB-1 and ErbB-2 Receptors

Oncostatin M Promotes STAT3 Activation, VEGF Production, and Invasion in Osteosarcoma Cell Lines