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Publications by Malin Bäckström

Novel Carbohydrate Binding Site Recognizing Blood Group a and B Determinants in a Hybrid of Cholera Toxin andEscherichia coliHeat-labile Enterotoxin B-Subunits

Journal of Biological Chemistry

English

Cervical Mucins Carry Α(1,2)fucosylated Glycans That Partly Protect From Experimental Vaginal Candidiasis

Glycoconjugate Journal

English

Related publications

Structural Basis for the Differential Toxicity of Cholera Toxin andEscherichia coliHeat-labile Enterotoxin

Journal of Biological Chemistry

English

The B Subunits of Cholera andEscherichia Coliheat-Labile Toxins Enhance the Immune Responses in Mice Orally Immunised With a Recombinant Live P-Fimbrial Vaccine for Avian pathogenicE. Coli

Acta Veterinaria Hungarica

Veterinary

2014

English

A Hybrid Polymer to Target Blood Group Dependence of Cholera Toxin

Organic and Biomolecular Chemistry

Organic Chemistry

Theoretical Chemistry

Biochemistry

Physical

2020

English

A Nontoxic Cholera Enterotoxin (CT) Analog Is Chimeric With Regard to Both Epitypes of CT-B Subunits, CT-B-1 and CT-B-2.

Infection and Immunity

English

Cell Clustering and Delay/Arrest in T-Cell Division Implicate a Novel Mechanism of Immune Modulation by E. Coli Heat-Labile Enterotoxin B-Subunits

Cellular Immunology

Immunology

2015

English

Bile and Unsaturated Fatty Acids Inhibit the Binding of Cholera Toxin and Escherichia Coli Heat-Labile Enterotoxin to GM1 Receptor

Antimicrobial Agents and Chemotherapy

Infectious Diseases

Pharmacology

2007

English

A Novel NF- B Binding Site Controls Human Granzyme B Gene Transcription

Journal of Immunology

Allergy

Immunology

2006

English

Intermolecular Interactions Between the a and B Subunits of Heat-Labile Enterotoxin From Escherichia Coli Promote Holotoxin Assembly and Stability in Vivo.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1992

English

Potent Immunogenicity of the B Subunits of Escherichia Coli Heat-Labile Enterotoxin: Receptor Binding Is Essential and Induces Differential Modulation of Lymphocyte Subsets.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1996

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Malin Bäckström

Novel Carbohydrate Binding Site Recognizing Blood Group a and B Determinants in a Hybrid of Cholera Toxin andEscherichia coliHeat-labile Enterotoxin B-Subunits

Cervical Mucins Carry Α(1,2)fucosylated Glycans That Partly Protect From Experimental Vaginal Candidiasis

Related publications

Structural Basis for the Differential Toxicity of Cholera Toxin andEscherichia coliHeat-labile Enterotoxin

The B Subunits of Cholera andEscherichia Coliheat-Labile Toxins Enhance the Immune Responses in Mice Orally Immunised With a Recombinant Live P-Fimbrial Vaccine for Avian pathogenicE. Coli

A Hybrid Polymer to Target Blood Group Dependence of Cholera Toxin

A Nontoxic Cholera Enterotoxin (CT) Analog Is Chimeric With Regard to Both Epitypes of CT-B Subunits, CT-B-1 and CT-B-2.

Cell Clustering and Delay/Arrest in T-Cell Division Implicate a Novel Mechanism of Immune Modulation by E. Coli Heat-Labile Enterotoxin B-Subunits

Bile and Unsaturated Fatty Acids Inhibit the Binding of Cholera Toxin and Escherichia Coli Heat-Labile Enterotoxin to GM1 Receptor

A Novel NF- B Binding Site Controls Human Granzyme B Gene Transcription

Intermolecular Interactions Between the a and B Subunits of Heat-Labile Enterotoxin From Escherichia Coli Promote Holotoxin Assembly and Stability in Vivo.

Potent Immunogenicity of the B Subunits of Escherichia Coli Heat-Labile Enterotoxin: Receptor Binding Is Essential and Induces Differential Modulation of Lymphocyte Subsets.