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Publications by Marcelo J. Kuroda

Simian Immunodeficiency Virus Targeting of CXCR3 + CD4 + T Cells in Secondary Lymphoid Organs Is Associated With Robust CXCL10 Expression in Monocyte/Macrophage Subsets

Journal of Virology

English

Expression of the Mannose Receptor CD206 in HIV and SIV Encephalitis: A Phenotypic Switch of Brain Perivascular Macrophages With Virus Infection

Journal of NeuroImmune Pharmacology

English

Increased Expression of CD169 on Blood Monocytes and Its Regulation by Virus and CD8 T Cells in Macaque Models of HIV Infection and AIDS

AIDS Research and Human Retroviruses

English

A Commonly Recognized Simian Immunodeficiency Virus Nef Epitope Presented to Cytotoxic T Lymphocytes of Indian-Origin Rhesus Monkeys by the Prevalent Major Histocompatibility Complex Class I Allele Mamu-A*02

English

Simian Immunodeficiency Virus Evades a Dominant Epitope-Specific Cytotoxic T Lymphocyte Response Through a Mutation Resulting in the Accelerated Dissociation of Viral Peptide and MHC Class I

Journal of Immunology

Allergy

Immunology

2000

English

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Standardization in Flow Cytometry: Correct Sample Handling as a Priority

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Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Marcelo J. Kuroda

Simian Immunodeficiency Virus Targeting of CXCR3 + CD4 + T Cells in Secondary Lymphoid Organs Is Associated With Robust CXCL10 Expression in Monocyte/Macrophage Subsets

Expression of the Mannose Receptor CD206 in HIV and SIV Encephalitis: A Phenotypic Switch of Brain Perivascular Macrophages With Virus Infection

Increased Expression of CD169 on Blood Monocytes and Its Regulation by Virus and CD8 T Cells in Macaque Models of HIV Infection and AIDS

A Commonly Recognized Simian Immunodeficiency Virus Nef Epitope Presented to Cytotoxic T Lymphocytes of Indian-Origin Rhesus Monkeys by the Prevalent Major Histocompatibility Complex Class I Allele Mamu-A*02

Simian Immunodeficiency Virus Evades a Dominant Epitope-Specific Cytotoxic T Lymphocyte Response Through a Mutation Resulting in the Accelerated Dissociation of Viral Peptide and MHC Class I

Related publications

COVID-19: Risk for Cytokine Targeting in Chronic Inflammatory Diseases?

Sterile Inflammation: Sensing and Reacting to Damage

DAMP-driven Metabolic Adaptation

The Regulation of IL-10 Production by Immune Cells

Complement as a Target in COVID-19?

The Function of Fcγ Receptors in Dendritic Cells and Macrophages

Monocyte Recruitment During Infection and Inflammation

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Heat Shock Proteins Are No DAMPs, Rather 'DAMPERs'

Standardization in Flow Cytometry: Correct Sample Handling as a Priority