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Publications by Maria Bonomelli
Pathophysiologic Implications of Reduced Podocyte Number in a Rat Model of Progressive Glomerular Injury
American Journal of Pathology
Forensic Medicine
Pathology
Related publications
Podocyte Loss and Progressive Glomerular Injury in Type II Diabetes.
Journal of Clinical Investigation
Medicine
Evaluation of a Thick and Thin Section Method for Estimation of Podocyte Number, Glomerular Volume, and Glomerular Volume Per Podocyte in Rat Kidney With Wilms' Tumor-1 Protein Used as a Podocyte Nuclear Marker
Journal of the American Society of Nephrology : JASN
Medicine
Nephrology
Mechanisms Underlying Transition From Acute Glomerular Injury to Late Glomerular Sclerosis in a Rat Model of Nephrotic Syndrome.
Journal of Clinical Investigation
Medicine
Indomethacin Administered Early in the Postnatal Period Results in Reduced Glomerular Number in the Adult Rat
American Journal of Physiology - Renal Physiology
Urology
Physiology
Induction of Podocyte-Derived VEGF Ameliorates Podocyte Injury and Subsequent Abnormal Glomerular Development Caused by Puromycin Aminonucleoside
Pediatric Research
Child Health
Pediatrics
Perinatology
Mesenchymal Stem Cells Ameliorate Podocyte Injury and Proteinuria in a Type 1 Diabetic Nephropathy Rat Model
Biology of Blood and Marrow Transplantation
Transplantation
Hematology
Podocyte-Specific RAP1GAP Expression Contributes to Focal Segmental Glomerulosclerosis–associated Glomerular Injury
Journal of Clinical Investigation
Medicine
Plasminogenuria Is Associated With Podocyte Injury, Edema, and Kidney Dysfunction in Incident Glomerular Disease
Hydrochloride Pioglitazone Protects Diabetic Rats Against Podocyte Injury Through Preserving Glomerular Podocalyxin Expression
Arquivos Brasileiros de Endocrinologia & Metabologia