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Publications by Martin Biel

The Murine HCN3 Gene Encodes a Hyperpolarization-Activated Cation Channel With Slow Kinetics and Unique Response to Cyclic Nucleotides

Journal of Biological Chemistry

English

Absence of the Γ Subunit of the Skeletal Muscle Dihydropyridine Receptor Increases L-Type Ca2+Currents and Alters Channel Inactivation Properties

Journal of Biological Chemistry

English

Primary Structure and Functional Expression of a High Voltage Activated Calcium Channel From Rabbit Lung

English

Morphological Characterization of the Retina of the CNGA3−/−Rho−/−Mutant Mouse Lacking Functional Cones and Rods

Investigative Ophthalmology and Visual Science

Molecular Neuroscience

English

The Two-Pore Channel TPCN2 Mediates NAADP-dependent Ca2+-Release From Lysosomal Stores

Pflugers Archiv European Journal of Physiology

Clinical Biochemistry

Physiology

2009

English

10 Cyclic Nucleotide Gated Channels

Advances in Second Messenger and Phosphoprotein Research

1999

English

Oita International Electrocardiology Symposium 2000 “Electrophysiology and Management of Lethal Arrhythmias in the New Millennium: From Genes to Bedside”

Japanese Journal of Electrocardiology

2000

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Martin Biel

The Murine HCN3 Gene Encodes a Hyperpolarization-Activated Cation Channel With Slow Kinetics and Unique Response to Cyclic Nucleotides

Absence of the Γ Subunit of the Skeletal Muscle Dihydropyridine Receptor Increases L-Type Ca2+Currents and Alters Channel Inactivation Properties

Primary Structure and Functional Expression of a High Voltage Activated Calcium Channel From Rabbit Lung

Morphological Characterization of the Retina of the CNGA3−/−Rho−/−Mutant Mouse Lacking Functional Cones and Rods

The Two-Pore Channel TPCN2 Mediates NAADP-dependent Ca2+-Release From Lysosomal Stores

10 Cyclic Nucleotide Gated Channels

Oita International Electrocardiology Symposium 2000 “Electrophysiology and Management of Lethal Arrhythmias in the New Millennium: From Genes to Bedside”