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Publications by Marwa Kamel
Erratum to “Overhydroxylation of Lysine of Collagen Increases Uterine Fibroids Proliferation: Roles of Lysyl Hydroxylases, Lysyl Oxidases, and Matrix Metalloproteinases”
BioMed Research International
Immunology
Molecular Biology
Biochemistry
Microbiology
Medicine
Genetics
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Figure 5: Multiple Alignment of Archaeal Lysyl Oxidases.
Differential Cleavage of Lysyl Oxidase by the Metalloproteinases BMP1 and ADAMTS2/14 Regulates Collagen Binding Through a Tyrosine Sulfate Domain
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The Lysyl Oxidases LOX and LOXL2 Are Necessary and Sufficient to Repress E-Cadherin in Hypoxia
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Proteolytic Processing of Lysyl Oxidase Like-2 in the Extracellular Matrix Is Required for Crosslinking of Basement Membrane Collagen IV
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Lysyl Hydroxylase 3 Is a Multifunctional Protein Possessing Collagen Glucosyltransferase Activity
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The Extracellular Matrix Contributes to Mechanotransduction in Uterine Fibroids
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Developing Functional Musculoskeletal Tissues Through Hypoxia and Lysyl Oxidase-Induced Collagen Cross-Linking
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Multidisciplinary
Lysyl Oxidase Activity Is Required for Ordered Collagen Fibrillogenesis by Tendon Cells
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