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Publications by Mary G Sorci-Thomas
Activation of Lecithin:cholesterol Acyltransferase by HDL ApoA-I Central Helices
Clinical Lipidology
Related publications
Dynamics of Activation of Lecithin:Cholesterol Acyltransferase by Apolipoprotein A-I
Biochemistry
Biochemistry
A Retractable Lid in Lecithin:cholesterol Acyltransferase Provides a Structural Mechanism for Activation by Apolipoprotein A-I
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
An apoA-I Mimetic Peptibody Generates HDL-like Particles and Increases Alpha-1 HDL Subfraction in Mice
Journal of Lipid Research
Biochemistry
Endocrinology
Cell Biology
Characterization of Subspecies of Apolipoprotein A-I-Containing Lipoprotein in Homozygotes for Familial Lecithin:cholesterol Acyltransferase Deficiency
Arteriosclerosis and Thrombosis: A Journal of Vascular Biology
N-Terminal Mutation of apoA-I and Interaction With ABCA1 Reveal Mechanisms of Nascent HDL Biogenesis
Journal of Lipid Research
Biochemistry
Endocrinology
Cell Biology
Direct Detection of ABCA1-dependent HDL Formation Based on Lipidation-Induced Hydrophobicity Change in apoA-I
Journal of Lipid Research
Biochemistry
Endocrinology
Cell Biology
Overexpression of Lecithin:cholesterol Acyltransferase in Transgenic Rabbits Prevents Diet-Induced Atherosclerosis.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
The -Chain of Cell Surface F0F1 ATPase Modulates ApoA-I and HDL Transcytosis Through Aortic Endothelial Cells
Arteriosclerosis, Thrombosis, and Vascular Biology
Cardiovascular Medicine
Cardiology
Naturally Occurring and Bioengineered apoA-I Mutations That Inhibit the Conversion of Discoidal to Spherical HDL: The Abnormal HDL Phenotypes Can Be Corrected by Treatment With LCAT
Biochemical Journal
Biochemistry
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