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Publications by Marylynn S. Barkley
Medroxyprogesterone Acetate Decreases Th1, Th17, and Increases Th22 Responses via AHR Signaling Which Could Affect Susceptibility to Infections and Inflammatory Disease
Frontiers in Immunology
Allergy
Immunology
Related publications
Role and Plasticity of Th1 and Th17 Responses in Immunity to Staphylococcus Aureus
Human Vaccines and Immunotherapeutics
Medicine
Allergy
Immunology
Pharmacology
Higher Susceptibility to Experimental Autoimmune Encephalomyelitis in Muc1-Deficient Mice Is Associated With Increased Th1/Th17 Responses
Brain, Behavior, and Immunity
Endocrine
Immunology
Behavioral Neuroscience
Autonomic Systems
Blockade of IL-10 Signaling During Bacillus Calmette-Guerin Vaccination Enhances and Sustains Th1, Th17, and Innate Lymphoid IFN- And IL-17 Responses and Increases Protection to Mycobacterium Tuberculosis Infection
Journal of Immunology
Allergy
Immunology
PDLIM2 Restricts Th1 and Th17 Differentiation and Prevents Autoimmune Disease
Cell and Bioscience
Biochemistry
Genetics
Molecular Biology
Subcutaneous Depo-Medroxyprogesterone Acetate
Journal of Family Planning and Reproductive Health Care
Murine Norovirus Infection Induces TH1 Inflammatory Responses to Dietary Antigens
Cell Host and Microbe
Immunology
Molecular Biology
Cancer Research
Virology
Microbiology
Microbiology
Parasitology
Regulation of IL-12p40 by HIF Controls Th1/Th17 Responses to Prevent Mucosal Inflammation
Mucosal Immunology
Allergy
Immunology
TGFβ1 Signaling Sustains Aryl Hydrocarbon Receptor (AHR) Expression and Restrains the Pathogenic Potential of TH17 Cells by an AHR-independent Mechanism
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Dopamine Receptor D3 Signaling on CD4+T Cells Favors Th1- And Th17-Mediated Immunity
Journal of Immunology
Allergy
Immunology
