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Publications by Matthew P McCormack

Myeloproliferative Disorder and Leukaemia in Mice Induced by Different Classes of Constitutive Mutants of the Human IL-3/IL-5/GM-CSF Receptor Common Β Subunit

Oncogene

English

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A Cell Type-Specific Constitutive Point Mutant of the Common Β-Subunit of the Human Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF), Interleukin (IL)-3, and IL-5 Receptors Requires the GM-CSF Receptor Α-Subunit for Activation

Journal of Biological Chemistry

English

Essential Mechanisms of Differential Activation of Eosinophils by IL-3 Compared to GM-CSF and IL-5

Critical Reviews in Immunology

Allergy

Immunology

2017

English

Extracellular Truncations of H Beta C, the Common Signaling Subunit for Interleukin-3 (IL-3), Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF), and IL-5, Lead to Ligand-Independent Activation

English

Slow-Dissociation Effect of Common Signaling Subunit Β on IL5 and GM-CSF Receptor Assembly

English

IL-3 Receptor Signaling Is Dispensable for BCR-ABL-induced Myeloproliferative Disease

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2003

English

Cytoplasmic Domains of the Human Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF) Receptor Β Chain (Hβc) Responsible for Human GM-CSF-induced Myeloid Cell Differentiation

Journal of Biological Chemistry

English

Toll‐like Receptor 4 Regulates Spontaneous Intestinal Tumorigenesis by Up‐regulating IL‐6 and GM‐CSF

Journal of Cellular and Molecular Medicine

Molecular Medicine

Cell Biology

2020

English

Multi-Colony Stimulating Activity of Interleukin 5 (IL-5) on Hematopoietic Progenitors From Transgenic Mice That Express IL-5 Receptor Alpha Subunit Constitutively

Journal of Experimental Medicine

English

Human IL-34 and CSF-1 Establish Structurally Similar Extracellular Assemblies With Their Common Hematopoietic Receptor

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Matthew P McCormack

Myeloproliferative Disorder and Leukaemia in Mice Induced by Different Classes of Constitutive Mutants of the Human IL-3/IL-5/GM-CSF Receptor Common Β Subunit

Related publications

A Cell Type-Specific Constitutive Point Mutant of the Common Β-Subunit of the Human Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF), Interleukin (IL)-3, and IL-5 Receptors Requires the GM-CSF Receptor Α-Subunit for Activation

Essential Mechanisms of Differential Activation of Eosinophils by IL-3 Compared to GM-CSF and IL-5

Extracellular Truncations of H Beta C, the Common Signaling Subunit for Interleukin-3 (IL-3), Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF), and IL-5, Lead to Ligand-Independent Activation

Slow-Dissociation Effect of Common Signaling Subunit Β on IL5 and GM-CSF Receptor Assembly

IL-3 Receptor Signaling Is Dispensable for BCR-ABL-induced Myeloproliferative Disease

Cytoplasmic Domains of the Human Granulocyte-Macrophage Colony-Stimulating Factor (GM-CSF) Receptor Β Chain (Hβc) Responsible for Human GM-CSF-induced Myeloid Cell Differentiation

Toll‐like Receptor 4 Regulates Spontaneous Intestinal Tumorigenesis by Up‐regulating IL‐6 and GM‐CSF

Multi-Colony Stimulating Activity of Interleukin 5 (IL-5) on Hematopoietic Progenitors From Transgenic Mice That Express IL-5 Receptor Alpha Subunit Constitutively

Human IL-34 and CSF-1 Establish Structurally Similar Extracellular Assemblies With Their Common Hematopoietic Receptor