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Publications by Mengying Wei
Semi‑random Mutagenesis Profile of BCR‑ABL During Imatinib Resistance Acquirement in K562 Cells
Molecular Medicine Reports
Oncology
Genetics
Molecular Biology
Biochemistry
Cancer Research
Molecular Medicine
Related publications
Imatinib Mesylate Resistance Through BCR-ABL Independence in Chronic Myelogenous Leukemia
Cancer Research
Cancer Research
Oncology
A Non-Atp-Competitive Inhibitor of BCR-ABL Overrides Imatinib Resistance
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Nilotinib Significantly Induces Apoptosis in Imatinib Resistant K562 Cells With Wild-Type BCR–ABL, as Effectively as in Parental Sensitive Counterparts
Hematology
Hematology
NPB001-05 Inhibits BCR-Abl Kinase Leading to Apoptosis of Imatinib-Resistant Cells
Frontiers in Bioscience - Elite
Immunology
Molecular Biology
Biochemistry
Microbiology
Medicine
Genetics
In Vitro and in Vivo Activity of SKI-606, a Novel SRC-Abl Inhibitor, Against Imatinib-Resistant BCR-Abl+ Neoplastic Cells
Cancer Research
Cancer Research
Oncology
Identification of Drug Combinations Containing Imatinib for Treatment of BCR-ABL+ Leukemias
PLoS ONE
Multidisciplinary
Detection of Single Nucleotide Insertion of BCR/ABL Region in Imatinib-Resistant Human Myelogenous Leukemia SR-1 Cells
Experimental & Molecular Medicine
Protein Kinase CKIIa Interacts With the BCR Moiety of BCR/Abl and Mediates Proliferation of BCR/Abl-Expressing Cells
Oncogene
Cancer Research
Genetics
Molecular Biology
The Chronic Myelocytic Cell Line K562 Contains a Breakpoint in BCR and Produces a Chimeric BCR/C-Abl Transcript.
Molecular and Cellular Biology
Cell Biology
Molecular Biology
