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Publications by Michael G Kharas
The SRC Homology-2 Protein SHB Modulates Focal Adhesion Kinase Signaling in a BCR-ABL Myeloproliferative Disorder Causing Accelerated Progression of Disease
Journal of Hematology and Oncology
Cancer Research
Oncology
Hematology
Molecular Biology
Related publications
IL-3 Receptor Signaling Is Dispensable for BCR-ABL-induced Myeloproliferative Disease
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
In Vitro Phosphorylation of the Focal Adhesion Targeting Domain of Focal Adhesion Kinase by SRC Kinase
Biochemistry
Biochemistry
Suppression of E-Protein Activity Interferes With the Development of BCR-ABL-mediated Myeloproliferative Disease
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Protein Kinase CKIIa Interacts With the BCR Moiety of BCR/Abl and Mediates Proliferation of BCR/Abl-Expressing Cells
Oncogene
Cancer Research
Genetics
Molecular Biology
BCR-ABL Tyrosine Kinase
Science-Business eXchange
Inhibitors of SRC and Focal Adhesion Kinase Promote Endocrine Specification
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
SRC Family Kinases Phosphorylate the BCR-Abl SH3-SH2 Region and Modulate BCR-Abl Transforming Activity
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Adoptive Immunotherapy of BCR-ABL-induced Chronic Myeloid Leukemia-Like Myeloproliferative Disease in a Murine Model
Blood
Biochemistry
Immunology
Cell Biology
Hematology
Deletion of the SRC Homology 3 Domain and C-Terminal Proline-Rich Sequences in BCR-Abl Prevents Abl Interactor 2 Degradation and Spontaneous Cell Migration and Impairs Leukemogenesis
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
