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Publications by Michael Schoen
Amyloid Beta Protein-Induced Zinc Sequestration Leads to Synaptic Loss via Dysregulation of the ProSAP2/Shank3 Scaffold
Molecular Neurodegeneration
Molecular Neuroscience
Neurology
Molecular Biology
Cellular
Phenotypic and Functional Analysis of SHANK3 Stop Mutations Identified in Individuals With ASD and/or ID
Molecular Autism
Psychiatry
Mental Health
Developmental Neuroscience
Developmental Biology
Molecular Biology
Related publications
NMDA Receptor Subunit Composition Determines Beta-Amyloid-Induced Neurodegeneration and Synaptic Loss
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
S-Nitrosylation Activates Cdk5 and Contributes to Synaptic Spine Loss Induced by -Amyloid Peptide
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Intracellular Accumulation of Amyloid-Beta – A Predictor for Synaptic Dysfunction and Neuron Loss in Alzheimer’s Disease
Frontiers in Aging Neuroscience
Aging
Cognitive Neuroscience
Alzheimer's Disease Risk Factor Pyk2 Mediates Amyloid-Β Induced Synaptic Dysfunction and Loss
Journal of Neuroscience
Neuroscience
Neurotoxicity of Amyloid -Protein: Synaptic and Network Dysfunction
Cold Spring Harbor perspectives in medicine
Biochemistry
Medicine
Genetics
Molecular Biology
Destruxin E Decreases Beta-Amyloid Generation by Reducing Colocalization of Beta-Amyloid-Cleaving Enzyme 1 and Beta-Amyloid Protein Precursor
Neurodegenerative Diseases
Neurology
Erythropoietin Prevents PC12 Cells From Beta-Amyloid-Induced Apoptosis via PI3K⁄Akt Pathway
Translational Neurodegeneration
Molecular Neuroscience
Neurology
Cellular
Cognitive Neuroscience
Presenilin 1 Transgene Addition to Amyloid Precursor Protein Overexpressing Transgenic Rats Increases Amyloid Beta 42 Levels and Results in Loss of Memory Retention
BMC Neuroscience
Neuroscience
Cellular
Molecular Neuroscience
Beta-Amyloid Disrupted Synaptic Vesicle Endocytosis in Cultured Hippocampal Neurons
Neuroscience
Neuroscience
