Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
Publications by Miguel Silva-Ramos
Inhibition of Cholinergic Neurotransmission by Β 3 -Adrenoceptors Depends on Adenosine Release and a 1 -Receptor Activation in Human and Rat Urinary Bladders
American Journal of Physiology - Renal Physiology
Urology
Physiology
Related publications
Adenosine Receptor Expression and Function in Rat Striatal Cholinergic Interneurons
British Journal of Pharmacology
Pharmacology
The Association of Arrestin-3 With the Human Lutropin/Choriogonadotropin Receptor Depends Mostly on Receptor Activation Rather Than on Receptor Phosphorylation
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Modeling Study of Three-Dimensional Structure of Human Β Adrenoceptors and Receptor-Antagonists Interactions
Folia Pharmacologica Japonica
Pharmacology
Pharmacological Characterization of Adenosine A1 and A2 Receptors in the Bladder: Evidence for a Modulatory Adenosine Tone Regulating Non-Adrenergic Non-Cholinergic Neurotransmission
British Journal of Pharmacology
Pharmacology
Platelet-Derived Growth Factor-Bb-Induced Human Smooth Muscle Cell Proliferation Depends on Basic FGF Release and FGFR-1 Activation
Circulation Research
Cardiovascular Medicine
Physiology
Cardiology
Functional Importance of Cholinergic and Purinergic Neurotransmission for Micturition Contraction in the Normal, Unanaesthetized Rat
British Journal of Pharmacology
Pharmacology
Cellular Mechanisms for Amyloid Β-Protein Activation of Rat Cholinergic Basal Forebrain Neurons
Journal of Neurophysiology
Neuroscience
Physiology
5-Ht Induces Temporomandibular Joint Nociception in Rats Through the Local Release of Inflammatory Mediators and Activation of Local Β Adrenoceptors
Pharmacology Biochemistry and Behavior
Pharmacology
Biochemistry
Clinical Biochemistry
Behavioral Neuroscience
Toxicology
Biological Psychiatry
Activation of Phospholipase C Β by Gβγ and Gαq Involves C-Terminal Rearrangement to Release Auto-Inhibition