Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
Publications by Mike D. BEECROFT
Incremental Ca2+ Mobilization by Inositol Trisphosphate Receptors Is Unlikely to Be Mediated by Their Desensitization or Regulation by Luminal or Cytosolic Ca2+
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Related publications
Ca2+-Calmodulin Inhibits Ca2+ Release Mediated by Type-1, -2 and -3 Inositol Trisphosphate Receptors
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Author Correction: Structural Basis for the Regulation of Inositol Trisphosphate Receptors by Ca2+ and IP3
Nature Structural and Molecular Biology
Structural Biology
Molecular Biology
Inositol 1,4,5-Trisphosphate-Induced Ca2+ Release Is Inhibited by Mitochondrial Depolarization
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Calcium Release in HSY Cells Conforms to a Steady-State Mechanism Involving Regulation of the Inositol 1,4,5-Trisphosphate Receptor Ca2+ Channel by Luminal [Ca2+]
Journal of Cell Biology
Medicine
Cell Biology
The Bell-Shaped Ca2+Dependence of the Inositol 1,4,5-Trisphosphate-Induced Ca2+Release Is Modulated by Ca2+/Calmodulin
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Regulation of the Miniature Plasma Membrane Ca2+ChannelIminby Inositol 1,4,5-Trisphosphate Receptors
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
The Clustering of Inositol 1,4,5-Trisphosphate (IP3) Receptors Is Triggered by IP3 Binding and Facilitated by Depletion of the Ca2+ Store
Journal of Pharmacological Sciences
Molecular Medicine
Pharmacology
Regulation by Ca2+and Inositol 1,4,5-Trisphosphate (Insp3) of Single Recombinant Type 3 Insp3Receptor Channels
Journal of General Physiology
Physiology
Different Receptors Use Inositol Trisphosphate to Mobilize Ca2+ From Different Intracellular Pools
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
