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Publications by O. A. Dina
TRPC1 and TRPC6 Channels Cooperate With TRPV4 to Mediate Mechanical Hyperalgesia and Nociceptor Sensitization
Journal of Neuroscience
Neuroscience
Stress Induces a Switch of Intracellular Signaling in Sensory Neurons in a Model of Generalized Pain
Journal of Neuroscience
Neuroscience
Related publications
TRPV4 Channels Mediate Cardiac Fibroblast Differentiation by Integrating Mechanical and Soluble Signals
Journal of Molecular and Cellular Cardiology
Cardiovascular Medicine
Molecular Biology
Cardiology
Heteromeric TRPV4/TRPC1 Channels Mediate Calcium-Sensing Receptor-Induced Relaxations and Nitric Oxide Production in Mesenteric Arteries: Comparative Study Using Wild-Type and TRPC1−/- Mice
Channels
Medicine
Biochemistry
Biophysics
Human Digital Meissner Corpuscles Display Immunoreactivity for the Multifunctional Ion Channels Trpc6 and Trpv4
Anatomical Record
Evolution
Ecology
Systematics
Anatomy
Histology
Behavior
Biotechnology
Lysophospholipases Cooperate to Mediate Lipid Homeostasis and Lysophospholipid Signaling
Journal of Lipid Research
Biochemistry
Endocrinology
Cell Biology
Figure 10: Cancer-Induced Mechanical Hyperalgesia and Its Reversal Using the Selective TRPV4 Antagonist HC-067047.
Activation of TRPV4 Channels Does Not Mediate Inversion of Neurovascular Coupling After SAH
Expression Levels of TRPC1 and TRPC6 Ion Channels Are Reciprocally Altered in Aging Rat Aorta: Implications for Age-Related Vasospastic Disorders
AGE
VEGF Regulates TRPC6 Channels in Podocytes
Nephrology Dialysis Transplantation
Medicine
Transplantation
Nephrology
Hydrogen Sulfide-Induced Mechanical Hyperalgesia and Allodynia Require Activation of Both Cav3.2 and TRPA1 Channels in Mice
British Journal of Pharmacology
Pharmacology