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Publications by P. Karkkainen

Promoter Polymorphisms -359t/C and -303a/G of the Catalytic Subunit P110 Gene of Human Phosphatidylinositol 3-Kinase Are Not Associated With Insulin Secretion or Insulin Sensitivity in Finnish Subjects

Diabetes Care

English

Related publications

The Tumour Necrosis Factor Alpha -238 G → a and -308 G → a Promoter Polymorphisms Are Not Associated With Insulin Sensitivity and Insulin Secretion in Young Healthy Relatives of Type II Diabetic Patients

English

Association of Insulin Sensitivity and Glucose Tolerance With the C.825C>T Variant of the G Protein Beta-3 Subunit Gene

Journal of Diabetes and its Complications

English

Phosphoinositide 3-Kinase Catalytic Subunit Deletion and Regulatory Subunit Deletion Have Opposite Effects on Insulin Sensitivity in Mice

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2005

English

Insulin Receptor Substrate Gene Polymorphisms Are Associated With Metabolic Syndrome but Not With Its Components

Journal of Diabetes Mellitus

2013

English

Heart Rate in Relation to Insulin Sensitivity and Insulin Secretion in Nondiabetic Subjects

English

Insulin Stimulation of the Fatty Acid Synthase Promoter Is Mediated by the Phosphatidylinositol 3-Kinase Pathway

Journal of Biological Chemistry

English

Body Weight, Not Insulin Sensitivity or Secretion, May Predict Spontaneous Weight Changes in Nondiabetic and Prediabetic Subjects: The RISC Study

English

Insulin Secretion and Insulin Sensitivity in Diabetic and Non-Diabetic Subjects With Hepatic Nuclear Factor-1alpha (Maturity-Onset Diabetes of the Young-3) Mutations

European Journal of Endocrinology

English

Circulating Retinol-Binding Protein-4, Insulin Sensitivity, Insulin Secretion, and Insulin Disposition Index in Obese and Nonobese Subjects

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by P. Karkkainen

Promoter Polymorphisms -359t/C and -303a/G of the Catalytic Subunit P110 Gene of Human Phosphatidylinositol 3-Kinase Are Not Associated With Insulin Secretion or Insulin Sensitivity in Finnish Subjects

Related publications

The Tumour Necrosis Factor Alpha -238 G → a and -308 G → a Promoter Polymorphisms Are Not Associated With Insulin Sensitivity and Insulin Secretion in Young Healthy Relatives of Type II Diabetic Patients

Association of Insulin Sensitivity and Glucose Tolerance With the C.825C>T Variant of the G Protein Beta-3 Subunit Gene

Phosphoinositide 3-Kinase Catalytic Subunit Deletion and Regulatory Subunit Deletion Have Opposite Effects on Insulin Sensitivity in Mice

Insulin Receptor Substrate Gene Polymorphisms Are Associated With Metabolic Syndrome but Not With Its Components

Heart Rate in Relation to Insulin Sensitivity and Insulin Secretion in Nondiabetic Subjects

Insulin Stimulation of the Fatty Acid Synthase Promoter Is Mediated by the Phosphatidylinositol 3-Kinase Pathway

Body Weight, Not Insulin Sensitivity or Secretion, May Predict Spontaneous Weight Changes in Nondiabetic and Prediabetic Subjects: The RISC Study

Insulin Secretion and Insulin Sensitivity in Diabetic and Non-Diabetic Subjects With Hepatic Nuclear Factor-1alpha (Maturity-Onset Diabetes of the Young-3) Mutations

Circulating Retinol-Binding Protein-4, Insulin Sensitivity, Insulin Secretion, and Insulin Disposition Index in Obese and Nonobese Subjects