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Publications by Paola Giglio
PKR and GCN2 Stress Kinases Promote an ER Stress-Independent eIF2α Phosphorylation Responsible for Calreticulin Exposure in Melanoma Cells
OncoImmunology
Oncology
Allergy
Immunology
S-Nitrosylation Affects TRAP1 Structure and ATPase Activity and Modulates Cell Response to Apoptotic Stimuli
Related publications
Aldo-Keto Reductases Protect Metastatic Melanoma From ER Stress-Independent Ferroptosis
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Enhanced Interaction Between Pseudokinase and Kinase Domains in Gcn2 Stimulates eIF2α Phosphorylation in Starved Cells
PLoS Genetics
Evolution
Ecology
Genetics
Molecular Biology
Cancer Research
Systematics
Behavior
Honokiol Confers Immunogenicity by Dictating Calreticulin Exposure, Activating ER Stress and Inhibiting Epithelial-To-Mesenchymal Transition
Molecular Oncology
Cancer Research
Medicine
Oncology
Genetics
Molecular Medicine
Conserved Intermolecular Salt Bridge Required for Activation of Protein Kinases PKR, GCN2, and PERK
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
ER Stress Signalling Through eIF2α and CHOP, but Not IRE1α, Attenuates Adipogenesis in Mice
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
eIF2α Phosphorylation Tips the Balance to Apoptosis During Osmotic Stress
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
788 ATF6-MFN2 Axis Regulates Mitochondrial Dynamics to Promote Melanoma Cell Survival Upon ER Stress
Journal of Investigative Dermatology
Biochemistry
Dermatology
Cell Biology
Molecular Biology
BiP and Calreticulin Form an Abundant Complex That Is Independent of Endoplasmic Reticulum Stress
Plant Cell
Plant Science
Cell Biology
Isochaihulactone-Induced DDIT3 Causes ER Stress-Perk Independent Apoptosis in Glioblastoma Multiforme Cells
Oncotarget
Oncology
