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Publications by Paramita Chakrabarty
Integrative Approach to Sporadic Alzheimer’s Disease: Deficiency of TYROBP in a Tauopathy Mouse Model Reduces C1q and Normalizes Clinical Phenotype While Increasing Spread and State of Phosphorylation of Tau
Molecular Psychiatry
Psychiatry
Molecular Neuroscience
Mental Health
Molecular Biology
Cellular
Inflammatory Pre-Conditioning Restricts the Seeded Induction of Α-Synuclein Pathology in Wild Type Mice
Molecular Neurodegeneration
Molecular Neuroscience
Neurology
Molecular Biology
Cellular
Related publications
Correction: Integrative Approach to Sporadic Alzheimer’s Disease: Deficiency of TYROBP in Cerebral Aβ Amyloidosis Mouse Normalizes Clinical Phenotype and Complement Subnetwork Molecular Pathology Without Reducing Aβ Burden
Molecular Psychiatry
Psychiatry
Molecular Neuroscience
Mental Health
Molecular Biology
Cellular
Analysis of Tau Phosphorylation and Truncation in a Mouse Model of Human Tauopathy
American Journal of Pathology
Forensic Medicine
Pathology
Human Tau Expression Reduces Adult Neurogenesis in a Mouse Model of Tauopathy
Neurobiology of Aging
Aging
Gerontology
Developmental Biology
Geriatrics
Neuroscience
Neurology
Pazopanib Reduces Phosphorylated Tau Levels and Alters Astrocytes in a Mouse Model of Tauopathy
Journal of Alzheimer's Disease
Gerontology
Clinical Psychology
Mental Health
Psychiatry
Geriatrics
Medicine
Neuroscience
Diaminothiazoles Modify Tau Phosphorylation and Improve the Tauopathy in Mouse Models
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Stabilizing the Retromer Complex in a Human Stem Cell Model of Alzheimer’s Disease Reduces TAU Phosphorylation Independently of Amyloid Precursor Protein
Stem Cell Reports
Biochemistry
Genetics
Developmental Biology
Cell Biology
Metformin Promotes Tau Aggregation and Exacerbates Abnormal Behavior in a Mouse Model of Tauopathy
Molecular Neurodegeneration
Molecular Neuroscience
Neurology
Molecular Biology
Cellular
Imaging of Tau Pathology in a Tauopathy Mouse Model and in Alzheimer Patients Compared to Normal Controls
Neuron
Neuroscience
Brain Gene Expression of a Sporadic (Icv-STZ Mouse) and a Familial Mouse Model (3xTg-Ad Mouse) of Alzheimer’s Disease
PLoS ONE
Multidisciplinary