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Publications by Patricia M. Kralik

Podocyte-Specific Overexpression of the Antioxidant Metallothionein Reduces Diabetic Nephropathy

Journal of the American Society of Nephrology : JASN

English

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Glomerular Mesangial Cell and Podocyte Injuries in Diabetic Nephropathy

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Loss of PTEN Promotes Podocyte Cytoskeletal Rearrangement, Aggravating Diabetic Nephropathy

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Mechanism of NHX1 Inhibiting TRPV5-Mediated Podocyte Injury in Diabetic Nephropathy

American Journal of Clinical and Experimental Medicine

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Islet Transplantation Promotes Podocyte Regeneration in a Model of Diabetic Nephropathy

Turkish Journal of Medical Sciences

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Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy

Journal of Diabetes Research

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The Number of Podocyte and Slit Diaphragm Is Decreased in Experimental Diabetic Nephropathy

Acta Cirurgica Brasileira

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Β-Arrestins Promote Podocyte Injury by Inhibition of Autophagy in Diabetic Nephropathy

Cell Death and Disease

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Rtn1a-Mediated Endoplasmic Reticulum Stress in Podocyte Injury and Diabetic Nephropathy

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Overexpression of Metallothionein in Pancreatic -Cells Reduces Streptozotocin-Induced DNA Damage and Diabetes

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