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Publications by Patrick G. Quinn

3′,5′-Cyclic Adenosine Monophosphate Response Element-Binding Protein and CCAAT Enhancer-Binding Protein Are Dispensable for Insulin Inhibition of Phosphoenolpyruvate Carboxykinase Transcription and for Its Synergistic Induction by Protein Kinase a and Glucocorticoids

Molecular Endocrinology

English

Related publications

Cyclic Adenosine Monophosphate Response Element–binding Protein 2 (CRTC2)

Science-Business eXchange

2008

English

Role of the Isoforms of CCAAT/Enhancer-binding Protein in the Initiation of Phosphoenolpyruvate Carboxykinase (GTP) Gene Transcription at Birth

Journal of Biological Chemistry

English

CCAAT/Enhancer-binding Protein-Β Regulates Interferon-Induced Transcription Through a Novel Element

Journal of Biological Chemistry

English

Cyclic Adenosine Monophosphate-Response Element-Binding Protein Mediates the Proangiogenic or Proinflammatory Activity of Gremlin

Arteriosclerosis, Thrombosis, and Vascular Biology

Cardiovascular Medicine

Cardiology

2014

English

Factors Affecting the Binding of [3H]adenosine 3′:5′-Cyclic Monophosphate to Protein Kinase From Bovine Adrenal Cortex

English

Cyclic‐AMP Response Element Binding Protein

English

Fornix-Dependent Induction of Hippocampal CCAAT Enhancer-Binding Protein Β and Δ Co-Localizes With Phosphorylated cAMP Response Element-Binding Protein and Accompanies Long-Term Memory Consolidation

Journal of Neuroscience

Neuroscience

2001

English

Protein Degradation in Hepatocyte Monolayers. Effects of Glucagon, Adenosine 3':5'-Cyclic Monophosphate and Insulin

English

Inhibition of P90 Ribosomal S6 Kinase-Mediated CCAAT/Enhancer-binding Protein Β Activation and Cyclooxygenase-2 Expression by Salicylate

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Patrick G. Quinn

3′,5′-Cyclic Adenosine Monophosphate Response Element-Binding Protein and CCAAT Enhancer-Binding Protein Are Dispensable for Insulin Inhibition of Phosphoenolpyruvate Carboxykinase Transcription and for Its Synergistic Induction by Protein Kinase a and Glucocorticoids

Related publications

Cyclic Adenosine Monophosphate Response Element–binding Protein 2 (CRTC2)

Role of the Isoforms of CCAAT/Enhancer-binding Protein in the Initiation of Phosphoenolpyruvate Carboxykinase (GTP) Gene Transcription at Birth

CCAAT/Enhancer-binding Protein-Β Regulates Interferon-Induced Transcription Through a Novel Element

Cyclic Adenosine Monophosphate-Response Element-Binding Protein Mediates the Proangiogenic or Proinflammatory Activity of Gremlin

Factors Affecting the Binding of [3H]adenosine 3′:5′-Cyclic Monophosphate to Protein Kinase From Bovine Adrenal Cortex

Cyclic‐AMP Response Element Binding Protein

Fornix-Dependent Induction of Hippocampal CCAAT Enhancer-Binding Protein Β and Δ Co-Localizes With Phosphorylated cAMP Response Element-Binding Protein and Accompanies Long-Term Memory Consolidation

Protein Degradation in Hepatocyte Monolayers. Effects of Glucagon, Adenosine 3':5'-Cyclic Monophosphate and Insulin

Inhibition of P90 Ribosomal S6 Kinase-Mediated CCAAT/Enhancer-binding Protein Β Activation and Cyclooxygenase-2 Expression by Salicylate