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Publications by Peter A. Antinozzi
Mitochondrial Metabolism Sets the Maximal Limit of Fuel-Stimulated Insulin Secretion in a Model Pancreatic Beta Cell
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Related publications
P2Y Purinergic Potentiation of Glucose-Induced Insulin Secretion and Pancreatic Beta-Cell Metabolism
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
The MDM2–p53–pyruvate Carboxylase Signalling Axis Couples Mitochondrial Metabolism to Glucose-Stimulated Insulin Secretion in Pancreatic Β-Cells
Nature Communications
Astronomy
Genetics
Molecular Biology
Biochemistry
Chemistry
Physics
Mitochondrial Metabolism of Pyruvate Is Essential for Regulating Glucose-Stimulated Insulin Secretion
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Sphingosine 1-Phosphate (S1P) Regulates Glucose-Stimulated Insulin Secretion in Pancreatic Beta Cells
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Hydrogen Peroxide Alters Mitochondrial Activation and Insulin Secretion in Pancreatic Beta Cells
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Pancreatic Beta Cell Selective Deletion of Mitofusins 1 and 2 (Mfn1 and Mfn2) Disrupts Mitochondrial Architecture and Abrogates Glucose-Stimulated Insulin Secretion in Vivo
Mitochondrial GTP Regulates Glucose-Stimulated Insulin Secretion
Cell Metabolism
Cell Biology
Molecular Biology
Physiology
Junctophilin 3 Expresses in Pancreatic Beta Cells and Is Required for Glucose-Stimulated Insulin Secretion
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
A Unifying Organ Model of Pancreatic Insulin Secretion
PLoS ONE
Multidisciplinary