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Publications by Peter E. Czabotar

Mutation to Bax Beyond the BH3 Domain Disrupts Interactions With Pro-Survival Proteins and Promotes Apoptosis

Journal of Biological Chemistry

Molecular Biology

English

Characterization of a Novel Venetoclax Resistance Mutation (BCL2 Phe104Ile) Observed in Follicular Lymphoma

British Journal of Haematology

English

Related publications

BH3-only Proteins in Apoptosis and Beyond: An Overview

Cancer Research

Molecular Biology

English

Contribution of BH3-domain and Transmembrane-Domain to the Activity and Interaction of the Pore-Forming BCL-2 Proteins Bok, Bak, and Bax

Scientific Reports

Multidisciplinary

English

Bax Crystal Structures Reveal How BH3 Domains Activate Bax and Nucleate Its Oligomerization to Induce Apoptosis

Molecular Biology

English

Proapoptotic Protein Bax Heterodimerizes With BCL-2 and Homodimerizes With Bax via a Novel Domain (BH3) Distinct From BH1 and BH2

Journal of Biological Chemistry

Molecular Biology

English

Loss of PDK4 Switches the Hepatic NF-κB/TNF Pathway From Pro-Survival to Pro-Apoptosis

English

Loss of Hus1 Sensitizes Cells to Etoposide-Induced Apoptosis by Regulating BH3-only Proteins

Cancer Research

Molecular Biology

English

Bax-Deficiency Promotes Drug Resistance and Oncogenic Transformation by Attenuating P53-Dependent Apoptosis

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

English

BAX and BAK Proteins Are Required for Cyclin-Dependent Kinase Inhibitory Drugs to Cause Apoptosis

Molecular Cancer Therapeutics

Cancer Research

English

PUMA- And Bax-Induced Autophagy Contributes to Apoptosis

Cell Death and Differentiation

Molecular Biology

English