Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Poren Kong

Phillyrin, a Natural Lignan, Attenuates Tumor Necrosis Factor Α-Mediated Insulin Resistance and Lipolytic Acceleration in 3t3-L1 Adipocytes

Planta Medica

Pharmaceutical Science

English

Related publications

Tumor Necrosis Factor Promotes Phosphorylation and Binding of Insulin Receptor Substrate 1 to Phosphatidylinositol 3-Kinase in 3t3-L1 Adipocytes

Journal of Biological Chemistry

English

Visfatin Is Involved in TNFα-mediated Insulin Resistance via an NAD+/Sirt1/PTP1B Pathway in 3t3-L1 Adipocytes

English

Effect of Insulin on Farnesyltransferase Activity in 3t3-L1 Adipocytes

Journal of Biological Chemistry

English

Cross-Talk Between the Platelet-Derived Growth Factor and the Insulin Signaling Pathways in 3t3-L1 Adipocytes

Journal of Biological Chemistry

English

MAP4K4 Gene Silencing in Human Skeletal Muscle Prevents Tumor Necrosis Factor-Α-Induced Insulin Resistance

Journal of Biological Chemistry

English

Stevioside Potentiates Insulin Sensitivity by Elevating Insulin-Stimulated Glucose Uptake in 3t3-L1 Adipocytes

The Open Conference Proceedings Journal

2013

English

Insulin and Insulin-Like Growth Factor 1 Stimulate the Phosphorylation on Tyrosine of a 160 kDa Cytosolic Protein in 3t3-L1 Adipocytes

English

Expression Profiling Identifies Genes That Continue to Respond to Insulin in Adipocytes Made Insulin-Resistant by Treatment With Tumor Necrosis Factor-Α

Journal of Biological Chemistry

English

Compartment-Specific Regulation of Phosphoinositide 3-Kinase by Platelet-Derived Growth Factor and Insulin in 3t3-L1 Adipocytes

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Poren Kong

Phillyrin, a Natural Lignan, Attenuates Tumor Necrosis Factor Α-Mediated Insulin Resistance and Lipolytic Acceleration in 3t3-L1 Adipocytes

Related publications

Tumor Necrosis Factor Promotes Phosphorylation and Binding of Insulin Receptor Substrate 1 to Phosphatidylinositol 3-Kinase in 3t3-L1 Adipocytes

Visfatin Is Involved in TNFα-mediated Insulin Resistance via an NAD+/Sirt1/PTP1B Pathway in 3t3-L1 Adipocytes

Effect of Insulin on Farnesyltransferase Activity in 3t3-L1 Adipocytes

Cross-Talk Between the Platelet-Derived Growth Factor and the Insulin Signaling Pathways in 3t3-L1 Adipocytes

MAP4K4 Gene Silencing in Human Skeletal Muscle Prevents Tumor Necrosis Factor-Α-Induced Insulin Resistance

Stevioside Potentiates Insulin Sensitivity by Elevating Insulin-Stimulated Glucose Uptake in 3t3-L1 Adipocytes

Insulin and Insulin-Like Growth Factor 1 Stimulate the Phosphorylation on Tyrosine of a 160 kDa Cytosolic Protein in 3t3-L1 Adipocytes

Expression Profiling Identifies Genes That Continue to Respond to Insulin in Adipocytes Made Insulin-Resistant by Treatment With Tumor Necrosis Factor-Α

Compartment-Specific Regulation of Phosphoinositide 3-Kinase by Platelet-Derived Growth Factor and Insulin in 3t3-L1 Adipocytes