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Publications by Prince T. Ampem
Pharmacological Evidence for a Role of the Transient Receptor Potential Canonical 3 (TRPC3) Channel in Endoplasmic Reticulum Stress-Induced Apoptosis of Human Coronary Artery Endothelial Cells
Vascular Pharmacology
Molecular Medicine
Physiology
Pharmacology
Related publications
Evidence of a Role for Fibroblast Transient Receptor Potential Canonical 3 Ca2+ Channel in Renal Fibrosis
Journal of the American Society of Nephrology : JASN
Medicine
Nephrology
Transient Receptor Potential Canonical 1 Channel Mediates the Mechanical Stress‑induced Epithelial‑mesenchymal Transition of Human Bronchial Epithelial (16HBE) Cells
International Journal of Molecular Medicine
Medicine
Genetics
Inhibition of Autophagy by 3‑MA Enhances Endoplasmic Reticulum Stress‑induced Apoptosis in Human Nasopharyngeal Carcinoma Cells
Oncology Letters
Cancer Research
Oncology
P85 Deficiency Protects -Cells From Endoplasmic Reticulum Stress-Induced Apoptosis
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Inhibition of Autophagy Using 3‑methyladenine Increases Cisplatin‑induced Apoptosis by Increasing Endoplasmic Reticulum Stress in U251 Human Glioma Cells
Molecular Medicine Reports
Oncology
Genetics
Molecular Biology
Biochemistry
Cancer Research
Molecular Medicine
Luteolin-Induced Apoptosis Through Activation of Endoplasmic Reticulum Stress Sensors in Pheochromocytoma Cells
Molecular Medicine Reports
Oncology
Genetics
Molecular Biology
Biochemistry
Cancer Research
Molecular Medicine
Prion Protein Protects Cancer Cells Against Endoplasmic Reticulum Stress Induced Apoptosis
Virologica Sinica
Role of Endoplasmic Reticulum Stress in Α-Tea Mediated TRAIL/DR5 Death Receptor Dependent Apoptosis
PLoS ONE
Multidisciplinary
Licochalcone A-Induced Human Bladder Cancer T24 Cells Apoptosis Triggered by Mitochondria Dysfunction and Endoplasmic Reticulum Stress
BioMed Research International
Immunology
Molecular Biology
Biochemistry
Microbiology
Medicine
Genetics
