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Publications by Q X Li
Β-Arrestins Promote Podocyte Injury by Inhibition of Autophagy in Diabetic Nephropathy
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
VPR-Induced Cell Cycle Arrest Is Conserved Among Primate Lentiviruses.
Journal of Virology
Insect Science
Immunology
Microbiology
Virology
Related publications
Long Noncoding RNA MEG3 Suppresses Podocyte Injury in Diabetic Nephropathy by Inactivating WNT/Β-Catenin Signaling
PeerJ
Genetics
Molecular Biology
Biochemistry
Biological Sciences
Medicine
Agricultural
Neuroscience
MicroRNA-27a Promotes Podocyte Injury via PPARγ-mediated Β-Catenin Activation in Diabetic Nephropathy
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Mechanism of NHX1 Inhibiting TRPV5-Mediated Podocyte Injury in Diabetic Nephropathy
American Journal of Clinical and Experimental Medicine
NOD2 Promotes Renal Injury by Exacerbating Inflammation and Podocyte Insulin Resistance in Diabetic Nephropathy
Kidney International
Nephrology
Rtn1a-Mediated Endoplasmic Reticulum Stress in Podocyte Injury and Diabetic Nephropathy
Scientific Reports
Multidisciplinary
Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy
Journal of Diabetes Research
Endocrinology
Metabolism
Diabetes
Autophagy in Diabetic Nephropathy
Journal of Endocrinology
Endocrinology
Metabolism
Diabetes
Astragaloside IV Protects Against Podocyte Injury via SERCA2-dependent ER Stress Reduction and AMPKα-regulated Autophagy Induction in Streptozotocin-Induced Diabetic Nephropathy
Scientific Reports
Multidisciplinary
Exosome Secreted From Adipose-Derived Stem Cells Attenuates Diabetic Nephropathy by Promoting Autophagy Flux and Inhibiting Apoptosis in Podocyte
Stem Cell Research and Therapy
Genetics
Cell Biology
Molecular Biology
Biochemistry
Medicine
Molecular Medicine