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Publications by R Carnicer
D: A Dystrophin-Dependent Loss of Neuronal Nitric Oxide Synthase in Atrial Fibrillation (Af) Promotes Electricalremodelling and Creates a Substrate for the Maintenance of Af
Heart
Cardiovascular Medicine
Cardiology
Related publications
Neuronal Nitric Oxide Synthase and Dystrophin-Deficient Muscular Dystrophy.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Downregulation of Endocardial Nitric Oxide Synthase Expression and Nitric Oxide Production in Atrial Fibrillation: Potential Mechanisms for Atrial Thrombosis and Stroke
Circulation
Cardiovascular Medicine
Physiology
Cardiology
Cardioversion of Atrial Fibrillation in ENGAGE AF-TIMI 48
Clinical Cardiology
Medicine
Cardiovascular Medicine
Cardiology
Structural Basis of Neuronal Nitric-Oxide Synthase Interaction With Dystrophin Repeats 16 and 17
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Neuronal Nitric Oxide Synthase as a Substrate for the Evolution of Pseudosexual Behaviour in a Parthenogenetic Whiptail Lizard
Journal of Neuroendocrinology
Molecular Neuroscience
Endocrine
Endocrinology
Autonomic Systems
Cellular
Metabolism
Diabetes
A Review of Rate Control in Atrial Fibrillation, and the Rationale and Protocol for the RATE-AF Trial
BMJ Open
Medicine
Patients’ Views About Screening for Atrial Fibrillation (AF): A Qualitative Study in Primary Care
BMJ Open
Medicine
L337H Mutant of Rat Neuronal Nitric Oxide Synthase Resembles Human Neuronal Nitric Oxide Synthase Toward Inhibitors
Journal of Medicinal Chemistry
Drug Discovery
Molecular Medicine
Oxidative Stress Creates a Unique, CaMKII-mediated Substrate for Atrial Fibrillation in Heart Failure
JCI insight
Medicine
