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Publications by R. Grumont
The Nuclear Factor- B and P53 Pathways Function Independently in Primary Cells and Transformed Fibroblasts Responding to Genotoxic Damage
Molecular Cancer Research
Cancer Research
Oncology
Molecular Biology
Related publications
The Central Nervous System-Restricted Transcription Factor Olig2 Opposes P53 Responses to Genotoxic Damage in Neural Progenitors and Malignant Glioma
Cancer Cell
Cancer Research
Oncology
Cell Biology
Genotoxic Stress/P53-Induced DNAJB9 Inhibits the Pro-Apoptotic Function of P53
Cell Death and Differentiation
Cell Biology
Molecular Biology
Flavopiridol Induces P53 via Initial Inhibition of Mdm2 and P21 And, Independently of P53, Sensitizes Apoptosis-Reluctant Cells to Tumor Necrosis Factor
Cancer Research
Cancer Research
Oncology
Arsenic Is Cytotoxic and Genotoxic to Primary Human Lung Cells
Mutation Research - Genetic Toxicology and Environmental Mutagenesis
Mutagenesis
Toxicology
Health
Genetics
Salicylates Inhibit Flavivirus Replication Independently of Blocking Nuclear Factor Kappa B Activation
Journal of Virology
Insect Science
Immunology
Microbiology
Virology
The Proteasome Inhibitor Carfilzomib Functions Independently of P53 to Induce Cytotoxicity and an Atypical NF- B Response in Chronic Lymphocytic Leukemia Cells
Clinical Cancer Research
Cancer Research
Oncology
Cancer Cells Suppress P53 in Adjacent Fibroblasts
Oncogene
Cancer Research
Genetics
Molecular Biology
A Mutant P53 Protein Is Required for Maintenance of the Transformed Phenotype in Cells Transformed With P53 Plus Ras cDNAs.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Genotoxic Damage in Non-Irradiated Cells: Contribution From the Bystander Effect
Radiation Protection Dosimetry
Nuclear Medicine
Radiology
Ultrasound Technology
Public Health
Radiation
Environmental
Radiological
Imaging
Medicine
Occupational Health
