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Publications by Ralph S. Pero

Coexpression of IL-5 and Eotaxin-2 in Mice Creates an Eosinophil-Dependent Model of Respiratory Inflammation With Characteristics of Severe Asthma

Journal of Immunology

Allergy

Immunology

2007

English

Post-Translational Tyrosine Nitration of Eosinophil Granule Toxins Mediated by Eosinophil Peroxidase

Journal of Biological Chemistry

English

Related publications

The Role of the Eosinophil-Selective Chemokine, Eotaxin, in Allergic and Non-Allergic Airways Inflammation

Memorias do Instituto Oswaldo Cruz

Medicine

Microbiology

1997

English

Benralizumab: A Unique IL-5 Inhibitor for Severe Asthma

Journal of Asthma and Allergy

English

Schizophyllum Commune Induces IL-17-mediated Neutrophilic Airway Inflammation in OVA-induced Asthma Model Mice

Scientific Reports

Multidisciplinary

2019

English

Use of Sputum Eosinophil Counts to Guide Management in Children With Severe Asthma

English

Loss of Extracellular Superoxide Dismutase Induces Severe IL-23-Mediated Skin Inflammation in Mice

Journal of Investigative Dermatology

English

Evaluation of the Characteristics of Asthma in Severe and Extremely Severe COPD

International Journal of COPD

English

Effects of Nebulized Dexamethasone on the Respiratory Microbiota and Mycobiota and Relative Equine Herpesvirus‐1, 2, 4, 5 in an Equine Model of Asthma

Journal of Veterinary Internal Medicine

Medicine

Veterinary

2019

English

Effect of Omalizumab on Lung Function and Eosinophil Levels in Adolescents With Moderate-To-Severe Allergic Asthma

Annals of Allergy, Asthma and Immunology

English

Eosinophil Recruitment Into the Respiratory Epithelium Following Antigenic Challenge in Hyper-IgE Mice Is Accompanied by Interleukin 5-Dependent Bronchial Hyperresponsiveness.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1995

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Ralph S. Pero

Coexpression of IL-5 and Eotaxin-2 in Mice Creates an Eosinophil-Dependent Model of Respiratory Inflammation With Characteristics of Severe Asthma

Post-Translational Tyrosine Nitration of Eosinophil Granule Toxins Mediated by Eosinophil Peroxidase

Related publications

The Role of the Eosinophil-Selective Chemokine, Eotaxin, in Allergic and Non-Allergic Airways Inflammation

Benralizumab: A Unique IL-5 Inhibitor for Severe Asthma

Schizophyllum Commune Induces IL-17-mediated Neutrophilic Airway Inflammation in OVA-induced Asthma Model Mice

Use of Sputum Eosinophil Counts to Guide Management in Children With Severe Asthma

Loss of Extracellular Superoxide Dismutase Induces Severe IL-23-Mediated Skin Inflammation in Mice

Evaluation of the Characteristics of Asthma in Severe and Extremely Severe COPD

Effects of Nebulized Dexamethasone on the Respiratory Microbiota and Mycobiota and Relative Equine Herpesvirus‐1, 2, 4, 5 in an Equine Model of Asthma

Effect of Omalizumab on Lung Function and Eosinophil Levels in Adolescents With Moderate-To-Severe Allergic Asthma

Eosinophil Recruitment Into the Respiratory Epithelium Following Antigenic Challenge in Hyper-IgE Mice Is Accompanied by Interleukin 5-Dependent Bronchial Hyperresponsiveness.