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Publications by Reda Z. Mahfouz

Leukemogenic Nucleophosmin Mutation Disrupts the Transcription Factor Hub Regulating Granulo-Monocytic Fates

Journal of Clinical Investigation
Medicine
2018English

Runx1 Regulation of Pu.1 Corepressor/Coactivator Exchange Identifies Specific Molecular Targets for Leukemia Differentiation Therapy

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2014English

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Breast Tumor Specific Mutation in GATA3 Affects Physiological Mechanisms Regulating Transcription Factor Turnover

BMC Cancer
Cancer ResearchOncologyGenetics
2014English

Converting Cell Fates: Generating Hematopoietic Stem Cellsde Novovia Transcription Factor Reprogramming

Annals of the New York Academy of Sciences
GeneticsMolecular BiologyBiochemistryNeuroscienceHistoryPhilosophy of Science
2016English

Knockout of the Transcription Factor NRF2 Disrupts Spermatogenesis in an Age-Dependent Manner

Free Radical Biology and Medicine
BiochemistryPhysiology
2010English

Notch Signaling Patterns Drosophila Mesodermal Segments by Regulating the bHLH Transcription Factor Twist

Development (Cambridge)
Developmental BiologyMolecular Biology
2004English

Suppression of Interneuron Programs and Maintenance of Selected Spinal Motor Neuron Fates by the Transcription Factor AML1/Runx1

International Journal of Developmental Neuroscience
Developmental NeuroscienceDevelopmental Biology
2008English

DNA Damage Disrupts the P14ARF-B23(Nucleophosmin) Interaction and Triggers a Transient Subnuclear Redistribution of P14ARF

Cancer Research
Cancer ResearchOncology
2005English

Human Histone Chaperone Nucleophosmin Enhances Acetylation-Dependent Chromatin Transcription

Molecular and Cellular Biology
Cell BiologyMolecular Biology
2005English

NSC348884, a Nucleophosmin Inhibitor Disrupts Oligomer Formation and Induces Apoptosis in Human Cancer Cells

Oncogene
Cancer ResearchGeneticsMolecular Biology
2008English

Enhanced Production of Vascular Endothelial Growth Factor by Human Monocytic Cells Stimulated With Endotoxin Through Transcription Factor SP-1

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