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Publications by Robert William Caldwell
P38 Mitogen-Activated Protein Kinase (MAPK) Increases Arginase Activity and Contributes to Endothelial Dysfunction in Corpora Cavernosa From Angiotensin-Ii-Treated Mice
Journal of Sexual Medicine
Gynecology
Reproductive Medicine
Urology
Obstetrics
Related publications
P38 Mitogen-Activated Protein Kinase (P38 MAPK; MAPK14)
Science-Business eXchange
P38 Mitogen-Activated Protein Kinase (P38 MAPK; MAPK14)
Science-Business eXchange
P38 Mitogen-Activated Protein Kinase Inhibition Ameliorates Angiotensin II-Induced Target Organ Damage
Hypertension
Internal Medicine
P38 Mitogen-Activated Protein Kinase (P38 MAPK; MAPK14); X-Box Binding Protein 1 (XBP1)
Science-Business eXchange
Glucose Deprivation Increases Tau Phosphorylation via P38 Mitogen-Activated Protein Kinase
Aging Cell
Aging
Cell Biology
Chronic P38 Mitogen-Activated Protein Kinase Inhibition Improves Vascular Function and Remodeling in Angiotensin II-dependent Hypertension
JRAAS - Journal of the Renin-Angiotensin-Aldosterone System
Internal Medicine
Endocrinology
Interleukin-17a Promotes Aortic Endothelial Cell Activation via Transcriptionally and Post-Translationally Activating P38 Mitogen-Activated Protein Kinase (MAPK) Pathway
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Doxycycline Hyclate Protects Lipopolysaccharide-Induced Endothelial Barrier Dysfunction by Inhibiting the Activation of P38 Mitogen-Activated Protein Kinase
Biological and Pharmaceutical Bulletin
Medicine
Pharmacology
Pharmaceutical Science
A New Phosphospecific Cell-Based ELISA for P42/P44 Mitogen-Activated Protein Kinase (MAPK), P38 MAPK, Protein Kinase B and cAMP-response-element-binding Protein
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology