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Publications by S Mullaly
Intraperitoneal Lipopolysaccharide-Induced Neutrophil Sequestration in the Lung Microvasculature Is Due to a Factor Produced in the Peritoneal Cavity
Critical Care
Critical Care
Intensive Care Medicine
Related publications
Phosphoinositide 3-Kinase Required for Lipopolysaccharide-Induced Transepithelial Neutrophil Trafficking in the Lung
European Respiratory Journal
Medicine
Pulmonary
Respiratory Medicine
Neutrophil Influx in Response to a Peritoneal Infection With Salmonella Is Delayed in Lipopolysaccharide-Binding Protein or CD14-Deficient Mice
Journal of Immunology
Allergy
Immunology
Interleukin-10 Gene Transfer to Peritoneal Mesothelial Cells Suppresses Peritoneal Dissemination of Gastric Cancer Cells Due to a Persistently High Concentration in the Peritoneal Cavity
Cancer Gene Therapy
Cancer Research
Molecular Medicine
Molecular Biology
Megakaryocytic Potentiating Factor and Mature Mesothelin Stimulate the Growth of a Lung Cancer Cell Line in the Peritoneal Cavity of Mice
PLoS ONE
Multidisciplinary
Talin1 Methylation Is Required for Neutrophil Infiltration and Lipopolysaccharide-Induced Lethality
Journal of Immunology
Allergy
Immunology
Endothelium-Derived Toll-Like Receptor-4 Is the Key Molecule in LPS-induced Neutrophil Sequestration Into Lungs
Journal of Clinical Investigation
Medicine
Lipopolysaccharide Binding Protein and CD14 Interaction Induces Tumor Necrosis Factor-Alpha Generation and Neutrophil Sequestration in Lungs After Intratracheal Endotoxin.
Circulation Research
Cardiovascular Medicine
Physiology
Cardiology
Interleukin-17 Stimulates Intraperitoneal Neutrophil Infiltration Through the Release of the Chemokine GROα From Peritoneal Mesothelial Cells
Critical Care
Critical Care
Intensive Care Medicine
Salbutamol Inhibits Lipopolysaccharide-Induced Inflammatory Responses in Rat Peritoneal Macrophages
Journal of Toxicological Sciences
Medicine
Toxicology
